Vasopressin V1a receptor is required for nucleocytoplasmic transport of mineralocorticoid receptor

We previously reported that a deficiency in the vasopressin V1a receptor (V1aR) results in type 4 renal tubular acidosis, which suggests that vasopressin exerts direct effects on the physiological actions of aldosterone. We investigated the role of vasopressin for nucleocytoplasmic transport of mine...

Ausführliche Beschreibung

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	American journal of physiology. Renal physiology 2012-10, Vol.303 (7), p.F1080-F1088
Hauptverfasser:	Hori, Kahori, Nagai, Takanori, Izumi, Yuichiro, Kimura, Miho, Hasuike, Yukiko, Nakayama, Yushi, Nanami, Masayoshi, Tokuyama, Masanori, Otaki, Yoshinaga, Kuragano, Takahiro, Kohda, Yukimasa, Obinata, Masuo, Kawahara, Katsumasa, Tanoue, Akito, Tomita, Kimio, Nakanishi, Takeshi, Nonoguchi, Hiroshi
Format:	Artikel
Sprache:	eng
Schlagworte:	11-beta-Hydroxysteroid Dehydrogenase Type 2 - genetics 11-beta-Hydroxysteroid Dehydrogenase Type 2 - metabolism Animals Antigens Cell Line Cell Nucleus - genetics Cell Nucleus - metabolism Cells Cytoplasm Cytoplasm - genetics Cytoplasm - metabolism GTPase-Activating Proteins - genetics GTPase-Activating Proteins - metabolism Kidney diseases Kidney Medulla - metabolism Mice Mice, Knockout Protein Transport - genetics Proteins Rats Receptors, Mineralocorticoid - genetics Receptors, Mineralocorticoid - metabolism Receptors, Vasopressin - genetics Receptors, Vasopressin - metabolism RNA Interference Rodents Vasopressins - metabolism
Online-Zugang:	Volltext
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!

Schreiben Sie den ersten Kommentar!