Overexpression of Romo1 Promotes Production of Reactive Oxygen Species and Invasiveness of Hepatic Tumor Cells

Background & Aims Chronic oxidative stress from reactive oxygen species (ROS) produced by the mitochondria promotes hepatocarcinogenesis and tumor progression. However, the exact mechanism by which mitochondrial ROS contributes to tumor cell invasion is not known. We investigated the role of ROS...

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Veröffentlicht in:	Gastroenterology (New York, N.Y. 1943) N.Y. 1943), 2012-10, Vol.143 (4), p.1084-1094.e7
Hauptverfasser:	Chung, Jin Sil, Park, SunHoo, Park, Seon Ho, Park, Eun–Ran, Cha, Pu–Hyeon, Kim, Bu–Yeo, Chung, Young Min, Woo, Seon Rang, Han, Chul Ju, Kim, Sang–Bum, Suh, Kyung–Suk, Jang, Ja–June, Lee, Kyoungbun, Choi, Dong Wook, Lee, Sora, Lee, Gi Young, Hahm, Ki Baik, Shin, Jung Ar, Kim, Byung Soo, Noh, Kyung Hee, Kim, Tae Woo, Lee, Kee–Ho, Yoo, Young Do
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biomarkers, Tumor - metabolism Carcinoma, Hepatocellular - metabolism Carcinoma, Hepatocellular - pathology Cell Line, Tumor Chemotherapy Resistance Chi-Square Distribution Disease-Free Survival Female Gastroenterology and Hepatology Humans Kaplan-Meier Estimate Liver Cancer Liver Neoplasms - metabolism Liver Neoplasms - pathology Male Membrane Proteins - metabolism Metastasis Mice Middle Aged Mitochondrial Proteins - metabolism Neoplasm Invasiveness Prognostic Factor Proportional Hazards Models Rats Reactive Oxygen Species - metabolism Risk Factors
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