Advanced glycation end products enhance reactive oxygen and nitrogen species generation in neutrophils in vitro

Increased oxidative stress (OS) in diabetes mellitus is one of the major factors leading to diabetic pathology. However, the mediators and mechanism that provoke OS in diabetes is not fully understood, and it is possible that accumulation of advanced glycation end products (AGEs) formed secondary to...

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Veröffentlicht in:	Molecular and cellular biochemistry 2012-02, Vol.361 (1-2), p.289-296
Hauptverfasser:	Bansal, Savita, Siddarth, Manushi, Chawla, Diwesh, Banerjee, Basu D., Madhu, S. V., Tripathi, Ashok K.
Format:	Artikel
Sprache:	eng
Schlagworte:	Biochemistry Biomedical and Life Sciences Carbonyl compounds Cardiology Diabetes Enzymes Glycation End Products, Advanced - pharmacology Glycation End Products, Advanced - physiology Humans Leukocytes Life Sciences Lipid Peroxidation Medical Biochemistry NADPH Oxidases - metabolism Neutrophils - enzymology Neutrophils - metabolism Neutrophils - physiology Nitric oxide Nitric Oxide - metabolism Nitric Oxide Synthase Type II - metabolism Nitrogen Oncology Oxidases Oxidative Stress Oxygen Pathology Protein Carbonylation Proteins Serum Albumin - chemistry Superoxides - metabolism
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