Targeting translocator protein protects against myocardial ischemia/reperfusion injury by alleviating mitochondrial dysfunction
Ischemic heart disease (IHD) remains a leading cause of mortalities worldwide, necessitating timely reperfusion to reduce acute mortality. Paradoxically, reperfusion can induce myocardial ischemia/reperfusion (I/R) injury, which is primarily characterized by mitochondrial dysfunction. Translocator p...
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Veröffentlicht in: | Experimental and therapeutic medicine 2024-09, Vol.28 (3), p.349, Article 349 |
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