Silibinin treatment protects human skin cells from UVB injury through upregulation of estrogen receptors

Ultraviolet B (UVB) from the sunlight is a major environmental cause for human skin damages, inducing cell death, inflammation, senescence and even carcinogenesis. The natural flavonoid silibinin, clinically used as liver protectant, has protective effects against UVB-caused skin injury in vivo and...

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Veröffentlicht in:	Journal of photochemistry and photobiology. B, Biology Biology, 2021-03, Vol.216, p.112147, Article 112147
Hauptverfasser:	Liu, Weiwei, Wang, Fang, Li, Can, Otkur, Wuxiyar, Hayashi, Toshihiko, Mizuno, Kazunori, Hattori, Shunji, Fujisaki, Hitomi, Onodera, Satoshi, Ikejima, Takashi
Format:	Artikel
Sprache:	eng
Schlagworte:	17β-Estradiol Agonists Apoptosis Carcinogenesis Carcinogens Cell activation Cell death Cell survival Damage Estrogen receptor α Estrogen receptor β Estrogen receptors Estrogens Fibroblasts Flavonoids HaCaT cells Human foreskin fibroblasts Keratinocytes Pretreatment Receptors Senescence Sex hormones Silibinin Skin Skin injuries Survival Transfection Ultraviolet B Ultraviolet radiation Xenoestrogens
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