Insulin receptor deletion in pancreatic acinar cells leads to loss of antimicrobial peptide secretion and reduced intestinal barrier function: Possible link between diabetes and severity of pancreatitis

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Veröffentlicht in:Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.] 2020-11, Vol.20, p.S11-S11
Hauptverfasser: Sanchez-Alvarez, R., Sugden, S., Sans, M., Williams, J., Bruce, J.
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container_title Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
container_volume 20
creator Sanchez-Alvarez, R.
Sugden, S.
Sans, M.
Williams, J.
Bruce, J.
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doi_str_mv 10.1016/j.pan.2020.07.382
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ispartof Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.], 2020-11, Vol.20, p.S11-S11
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source Alma/SFX Local Collection
subjects Acinar cells
Antimicrobial agents
Antimicrobial peptides
Bacteria
Clonal deletion
Colon
Diabetes
Diabetes mellitus
Inflammation
Insulin
Intestine
Pancreas
Pancreatitis
Peptides
Rodents
Sepsis
title Insulin receptor deletion in pancreatic acinar cells leads to loss of antimicrobial peptide secretion and reduced intestinal barrier function: Possible link between diabetes and severity of pancreatitis
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