Insulin receptor deletion in pancreatic acinar cells leads to loss of antimicrobial peptide secretion and reduced intestinal barrier function: Possible link between diabetes and severity of pancreatitis
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Veröffentlicht in: | Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.] 2020-11, Vol.20, p.S11-S11 |
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container_title | Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.] |
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creator | Sanchez-Alvarez, R. Sugden, S. Sans, M. Williams, J. Bruce, J. |
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doi_str_mv | 10.1016/j.pan.2020.07.382 |
format | Article |
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ispartof | Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.], 2020-11, Vol.20, p.S11-S11 |
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language | eng |
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source | Alma/SFX Local Collection |
subjects | Acinar cells Antimicrobial agents Antimicrobial peptides Bacteria Clonal deletion Colon Diabetes Diabetes mellitus Inflammation Insulin Intestine Pancreas Pancreatitis Peptides Rodents Sepsis |
title | Insulin receptor deletion in pancreatic acinar cells leads to loss of antimicrobial peptide secretion and reduced intestinal barrier function: Possible link between diabetes and severity of pancreatitis |
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