RELATIONSHIP BETWEEN THE ENVIRONMENTAL ENDOCRINE DISRUPTOR BISPHENOL A AND DYSLIPIDEMIA: A FIVE-YEAR PROSPECTIVE STUDY
To investigate whether serum bisphenol A (BPA) concentration is related to the occurrence of dyslipidemia. A total of 574 adults were enrolled at baseline and followed up for 5 years. Concentrations of serum BPA, triglycerides (TGs), low-density lipoprotein (LDL) cholesterol, and high-density lipopr...
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| Veröffentlicht in: | Endocrine practice 2020-04, Vol.26 (4), p.399-406 |
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| creator | Li, Ruolin Yang, Shumin Gao, Rufei Deng, Yin Liu, Jiahuan Yuan, Chao Yao, Qingmei Lv, Xinke Wang, Kanran Ye, Xiaoqi Peng, Bin Hu, Jinbo Chen, Aijun |
| description | To investigate whether serum bisphenol A (BPA) concentration is related to the occurrence of dyslipidemia.
A total of 574 adults were enrolled at baseline and followed up for 5 years. Concentrations of serum BPA, triglycerides (TGs), low-density lipoprotein (LDL) cholesterol, and high-density lipoprotein (HDL) cholesterol were measured. Dyslipidemia was defined as the existence of one or more of the following conditions: high-LDL-cholesterolemia (LDL ≥140 mg/dL), hypertriglyceridemia (TGs ≥150 mg/dL), or low-HDL-cholesterolemia (HDL |
| doi_str_mv | 10.4158/EP-2019-0384 |
| format | Article |
| fullrecord | <record><control><sourceid>proquest_cross</sourceid><recordid>TN_cdi_proquest_journals_2389778193</recordid><sourceformat>XML</sourceformat><sourcesystem>PC</sourcesystem><sourcerecordid>2389778193</sourcerecordid><originalsourceid>FETCH-LOGICAL-c319t-18a23f0a49769bee6a3e8cb0e7b4f09e5c05b5482d502237a178ac5a2009fd9f3</originalsourceid><addsrcrecordid>eNo9kE1PwkAQhjdGI4jePJtNvFrdj5bueit0sZuUbdMuKKemLdtEIoItmPjvXQJ6mpk3T2YmDwC3GD262GNPInUIwtxBlLlnoI85dR3iInpue48ih3H81gNXXbdCiCCO2SXoUcyHDHPcB9-ZiAMtE5VHMoUjoV-FUFBHAgo1l1mipkLpILZTmIwzqQQMZZ7NUp1kcCTzNBIqiWEAAxXCcJHHMpWhmMrg2WYTORfOQgQZTLMkT8VY2wDmehYursFFU3505uZUB2A2EXocOXHyIsdB7NT2xZ2DWUlog0qX-0NeGTMsqWF1hYxfuQ3ixquRV3kuI0sPEUL9EvusrL2SIMSbJW_oANwf927bzdfedLtitdm3n_ZkQSjjvm8tUEs9HKm63XRda5pi276vy_anwKg4SC5EWhwkFwfJFr87Ld1Xa7P8h_-s0l-bs2zM</addsrcrecordid><sourcetype>Aggregation Database</sourcetype><iscdi>true</iscdi><recordtype>article</recordtype><pqid>2389778193</pqid></control><display><type>article</type><title>RELATIONSHIP BETWEEN THE ENVIRONMENTAL ENDOCRINE DISRUPTOR BISPHENOL A AND DYSLIPIDEMIA: A FIVE-YEAR PROSPECTIVE STUDY</title><source>MEDLINE</source><source>ProQuest Central UK/Ireland</source><source>Alma/SFX Local Collection</source><creator>Li, Ruolin ; Yang, Shumin ; Gao, Rufei ; Deng, Yin ; Liu, Jiahuan ; Yuan, Chao ; Yao, Qingmei ; Lv, Xinke ; Wang, Kanran ; Ye, Xiaoqi ; Peng, Bin ; Hu, Jinbo ; Chen, Aijun</creator><creatorcontrib>Li, Ruolin ; Yang, Shumin ; Gao, Rufei ; Deng, Yin ; Liu, Jiahuan ; Yuan, Chao ; Yao, Qingmei ; Lv, Xinke ; Wang, Kanran ; Ye, Xiaoqi ; Peng, Bin ; Hu, Jinbo ; Chen, Aijun</creatorcontrib><description>To investigate whether serum bisphenol A (BPA) concentration is related to the occurrence of dyslipidemia.
A total of 574 adults were enrolled at baseline and followed up for 5 years. Concentrations of serum BPA, triglycerides (TGs), low-density lipoprotein (LDL) cholesterol, and high-density lipoprotein (HDL) cholesterol were measured. Dyslipidemia was defined as the existence of one or more of the following conditions: high-LDL-cholesterolemia (LDL ≥140 mg/dL), hypertriglyceridemia (TGs ≥150 mg/dL), or low-HDL-cholesterolemia (HDL <40 mg/dL). Participants were stratified into tertiles according to low, median, and high baseline serum BPA levels. Multivariable linear and logistic regression models were used. Data from baseline and follow-up were used for cross-sectional and longitudinal analyses, respectively.
In the cross-sectional analysis, compared to subjects in the low BPA tertile, those in the high BPA tertile showed a higher level of LDL cholesterol (108.1 ± 24.4 mg/dL versus 119.5 ± 26.9 mg/dL;
<.05) and a lower level of HDL cholesterol (46.2 ± 11.7 mg/dL versus 39.5 ± 7.5 mg/dL;
<.05). In multivariable linear regression models, Z-transformed BPA was positively associated with LDL cholesterol (β= 0.13,
= .002) and negatively associated with HDL cholesterol (β= -0.28;
<.001). After cross-sectionally adjusting for confounders, subjects in higher BPA exposure was associated with a higher prevalence of low-HDL-cholesterolemia. Longitudinally, in subjects without low-HDL-cholesterolemia at baseline, each SD increment in baseline BPA was associated with a higher incidence of low-HDL-cholesterolemia after adjustment for confounders (odds ratio [95% confidence interval; CI] 2.76, 95% CI 1.21, 6.29).
Cross-sectionally, higher BPA exposure is associated with a higher prevalence of low-HDL-cholesterolemia. Longitudinally, baseline BPA is an independent predictor of the 5-year incidence of low-HDL-cholesterolemia.
= body mass index;
= bisphenol A;
= confidence interval;
= cardiovascular disease;
= environment, inflammation and metabolic diseases study;
= high density lipoprotein;
= low density lipoprotein;
= odds ratio;
= peroxisome proliferator-activated receptor;
= systolic blood pressure;
= triglyceride;
= Z-transformed bisphenol A.</description><identifier>ISSN: 1530-891X</identifier><identifier>EISSN: 1934-2403</identifier><identifier>DOI: 10.4158/EP-2019-0384</identifier><identifier>PMID: 31968191</identifier><language>eng</language><publisher>United States: Elsevier Limited</publisher><subject>Age ; Benzhydryl Compounds - adverse effects ; Bisphenol A ; Blood pressure ; Cardiovascular disease ; Cholesterol ; Cholesterol, HDL ; Chronic illnesses ; Cross-Sectional Studies ; Diabetes ; Dyslipidemias - chemically induced ; Endocrine Disruptors ; Exercise ; Family medical history ; Fasting ; Humans ; Hypertension ; Kidney diseases ; Lipids ; Low density lipoprotein ; Metabolic disorders ; Phenols - adverse effects ; Physical fitness ; Prospective Studies ; Risk Factors ; Studies ; Triglycerides ; Variables</subject><ispartof>Endocrine practice, 2020-04, Vol.26 (4), p.399-406</ispartof><rights>Copyright Allen Press Publishing Services Apr 2020</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c319t-18a23f0a49769bee6a3e8cb0e7b4f09e5c05b5482d502237a178ac5a2009fd9f3</citedby><cites>FETCH-LOGICAL-c319t-18a23f0a49769bee6a3e8cb0e7b4f09e5c05b5482d502237a178ac5a2009fd9f3</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktohtml>$$Uhttps://www.proquest.com/docview/2389778193?pq-origsite=primo$$EHTML$$P50$$Gproquest$$H</linktohtml><link.rule.ids>314,777,781,27905,27906,64364,64368,72218</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/31968191$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Li, Ruolin</creatorcontrib><creatorcontrib>Yang, Shumin</creatorcontrib><creatorcontrib>Gao, Rufei</creatorcontrib><creatorcontrib>Deng, Yin</creatorcontrib><creatorcontrib>Liu, Jiahuan</creatorcontrib><creatorcontrib>Yuan, Chao</creatorcontrib><creatorcontrib>Yao, Qingmei</creatorcontrib><creatorcontrib>Lv, Xinke</creatorcontrib><creatorcontrib>Wang, Kanran</creatorcontrib><creatorcontrib>Ye, Xiaoqi</creatorcontrib><creatorcontrib>Peng, Bin</creatorcontrib><creatorcontrib>Hu, Jinbo</creatorcontrib><creatorcontrib>Chen, Aijun</creatorcontrib><title>RELATIONSHIP BETWEEN THE ENVIRONMENTAL ENDOCRINE DISRUPTOR BISPHENOL A AND DYSLIPIDEMIA: A FIVE-YEAR PROSPECTIVE STUDY</title><title>Endocrine practice</title><addtitle>Endocr Pract</addtitle><description>To investigate whether serum bisphenol A (BPA) concentration is related to the occurrence of dyslipidemia.
A total of 574 adults were enrolled at baseline and followed up for 5 years. Concentrations of serum BPA, triglycerides (TGs), low-density lipoprotein (LDL) cholesterol, and high-density lipoprotein (HDL) cholesterol were measured. Dyslipidemia was defined as the existence of one or more of the following conditions: high-LDL-cholesterolemia (LDL ≥140 mg/dL), hypertriglyceridemia (TGs ≥150 mg/dL), or low-HDL-cholesterolemia (HDL <40 mg/dL). Participants were stratified into tertiles according to low, median, and high baseline serum BPA levels. Multivariable linear and logistic regression models were used. Data from baseline and follow-up were used for cross-sectional and longitudinal analyses, respectively.
In the cross-sectional analysis, compared to subjects in the low BPA tertile, those in the high BPA tertile showed a higher level of LDL cholesterol (108.1 ± 24.4 mg/dL versus 119.5 ± 26.9 mg/dL;
<.05) and a lower level of HDL cholesterol (46.2 ± 11.7 mg/dL versus 39.5 ± 7.5 mg/dL;
<.05). In multivariable linear regression models, Z-transformed BPA was positively associated with LDL cholesterol (β= 0.13,
= .002) and negatively associated with HDL cholesterol (β= -0.28;
<.001). After cross-sectionally adjusting for confounders, subjects in higher BPA exposure was associated with a higher prevalence of low-HDL-cholesterolemia. Longitudinally, in subjects without low-HDL-cholesterolemia at baseline, each SD increment in baseline BPA was associated with a higher incidence of low-HDL-cholesterolemia after adjustment for confounders (odds ratio [95% confidence interval; CI] 2.76, 95% CI 1.21, 6.29).
Cross-sectionally, higher BPA exposure is associated with a higher prevalence of low-HDL-cholesterolemia. Longitudinally, baseline BPA is an independent predictor of the 5-year incidence of low-HDL-cholesterolemia.
= body mass index;
= bisphenol A;
= confidence interval;
= cardiovascular disease;
= environment, inflammation and metabolic diseases study;
= high density lipoprotein;
= low density lipoprotein;
= odds ratio;
= peroxisome proliferator-activated receptor;
= systolic blood pressure;
= triglyceride;
= Z-transformed bisphenol A.</description><subject>Age</subject><subject>Benzhydryl Compounds - adverse effects</subject><subject>Bisphenol A</subject><subject>Blood pressure</subject><subject>Cardiovascular disease</subject><subject>Cholesterol</subject><subject>Cholesterol, HDL</subject><subject>Chronic illnesses</subject><subject>Cross-Sectional Studies</subject><subject>Diabetes</subject><subject>Dyslipidemias - chemically induced</subject><subject>Endocrine Disruptors</subject><subject>Exercise</subject><subject>Family medical history</subject><subject>Fasting</subject><subject>Humans</subject><subject>Hypertension</subject><subject>Kidney diseases</subject><subject>Lipids</subject><subject>Low density lipoprotein</subject><subject>Metabolic disorders</subject><subject>Phenols - adverse effects</subject><subject>Physical fitness</subject><subject>Prospective Studies</subject><subject>Risk Factors</subject><subject>Studies</subject><subject>Triglycerides</subject><subject>Variables</subject><issn>1530-891X</issn><issn>1934-2403</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2020</creationdate><recordtype>article</recordtype><sourceid>EIF</sourceid><sourceid>ABUWG</sourceid><sourceid>AFKRA</sourceid><sourceid>AZQEC</sourceid><sourceid>BENPR</sourceid><sourceid>CCPQU</sourceid><recordid>eNo9kE1PwkAQhjdGI4jePJtNvFrdj5bueit0sZuUbdMuKKemLdtEIoItmPjvXQJ6mpk3T2YmDwC3GD262GNPInUIwtxBlLlnoI85dR3iInpue48ih3H81gNXXbdCiCCO2SXoUcyHDHPcB9-ZiAMtE5VHMoUjoV-FUFBHAgo1l1mipkLpILZTmIwzqQQMZZ7NUp1kcCTzNBIqiWEAAxXCcJHHMpWhmMrg2WYTORfOQgQZTLMkT8VY2wDmehYursFFU3505uZUB2A2EXocOXHyIsdB7NT2xZ2DWUlog0qX-0NeGTMsqWF1hYxfuQ3ixquRV3kuI0sPEUL9EvusrL2SIMSbJW_oANwf927bzdfedLtitdm3n_ZkQSjjvm8tUEs9HKm63XRda5pi276vy_anwKg4SC5EWhwkFwfJFr87Ld1Xa7P8h_-s0l-bs2zM</recordid><startdate>202004</startdate><enddate>202004</enddate><creator>Li, Ruolin</creator><creator>Yang, Shumin</creator><creator>Gao, Rufei</creator><creator>Deng, Yin</creator><creator>Liu, Jiahuan</creator><creator>Yuan, Chao</creator><creator>Yao, Qingmei</creator><creator>Lv, Xinke</creator><creator>Wang, Kanran</creator><creator>Ye, Xiaoqi</creator><creator>Peng, Bin</creator><creator>Hu, Jinbo</creator><creator>Chen, Aijun</creator><general>Elsevier Limited</general><scope>CGR</scope><scope>CUY</scope><scope>CVF</scope><scope>ECM</scope><scope>EIF</scope><scope>NPM</scope><scope>AAYXX</scope><scope>CITATION</scope><scope>3V.</scope><scope>7RV</scope><scope>7X7</scope><scope>7XB</scope><scope>88C</scope><scope>88E</scope><scope>8FI</scope><scope>8FJ</scope><scope>8FK</scope><scope>ABUWG</scope><scope>AFKRA</scope><scope>AZQEC</scope><scope>BENPR</scope><scope>CCPQU</scope><scope>FYUFA</scope><scope>GHDGH</scope><scope>K9-</scope><scope>K9.</scope><scope>KB0</scope><scope>M0R</scope><scope>M0S</scope><scope>M0T</scope><scope>M1P</scope><scope>NAPCQ</scope><scope>PQEST</scope><scope>PQQKQ</scope><scope>PQUKI</scope><scope>PRINS</scope></search><sort><creationdate>202004</creationdate><title>RELATIONSHIP BETWEEN THE ENVIRONMENTAL ENDOCRINE DISRUPTOR BISPHENOL A AND DYSLIPIDEMIA: A FIVE-YEAR PROSPECTIVE STUDY</title><author>Li, Ruolin ; Yang, Shumin ; Gao, Rufei ; Deng, Yin ; Liu, Jiahuan ; Yuan, Chao ; Yao, Qingmei ; Lv, Xinke ; Wang, Kanran ; Ye, Xiaoqi ; Peng, Bin ; Hu, Jinbo ; Chen, Aijun</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-c319t-18a23f0a49769bee6a3e8cb0e7b4f09e5c05b5482d502237a178ac5a2009fd9f3</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2020</creationdate><topic>Age</topic><topic>Benzhydryl Compounds - adverse effects</topic><topic>Bisphenol A</topic><topic>Blood pressure</topic><topic>Cardiovascular disease</topic><topic>Cholesterol</topic><topic>Cholesterol, HDL</topic><topic>Chronic illnesses</topic><topic>Cross-Sectional Studies</topic><topic>Diabetes</topic><topic>Dyslipidemias - chemically induced</topic><topic>Endocrine Disruptors</topic><topic>Exercise</topic><topic>Family medical history</topic><topic>Fasting</topic><topic>Humans</topic><topic>Hypertension</topic><topic>Kidney diseases</topic><topic>Lipids</topic><topic>Low density lipoprotein</topic><topic>Metabolic disorders</topic><topic>Phenols - adverse effects</topic><topic>Physical fitness</topic><topic>Prospective Studies</topic><topic>Risk Factors</topic><topic>Studies</topic><topic>Triglycerides</topic><topic>Variables</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Li, Ruolin</creatorcontrib><creatorcontrib>Yang, Shumin</creatorcontrib><creatorcontrib>Gao, Rufei</creatorcontrib><creatorcontrib>Deng, Yin</creatorcontrib><creatorcontrib>Liu, Jiahuan</creatorcontrib><creatorcontrib>Yuan, Chao</creatorcontrib><creatorcontrib>Yao, Qingmei</creatorcontrib><creatorcontrib>Lv, Xinke</creatorcontrib><creatorcontrib>Wang, Kanran</creatorcontrib><creatorcontrib>Ye, Xiaoqi</creatorcontrib><creatorcontrib>Peng, Bin</creatorcontrib><creatorcontrib>Hu, Jinbo</creatorcontrib><creatorcontrib>Chen, Aijun</creatorcontrib><collection>Medline</collection><collection>MEDLINE</collection><collection>MEDLINE (Ovid)</collection><collection>MEDLINE</collection><collection>MEDLINE</collection><collection>PubMed</collection><collection>CrossRef</collection><collection>ProQuest Central (Corporate)</collection><collection>Nursing & Allied Health Database</collection><collection>Health & Medical Collection</collection><collection>ProQuest Central (purchase pre-March 2016)</collection><collection>Healthcare Administration Database (Alumni)</collection><collection>Medical Database (Alumni Edition)</collection><collection>Hospital Premium Collection</collection><collection>Hospital Premium Collection (Alumni Edition)</collection><collection>ProQuest Central (Alumni) (purchase pre-March 2016)</collection><collection>ProQuest Central (Alumni Edition)</collection><collection>ProQuest Central UK/Ireland</collection><collection>ProQuest Central Essentials</collection><collection>ProQuest Central</collection><collection>ProQuest One Community College</collection><collection>Health Research Premium Collection</collection><collection>Health Research Premium Collection (Alumni)</collection><collection>Consumer Health Database (Alumni Edition)</collection><collection>ProQuest Health & Medical Complete (Alumni)</collection><collection>Nursing & Allied Health Database (Alumni Edition)</collection><collection>Consumer Health Database</collection><collection>Health & Medical Collection (Alumni Edition)</collection><collection>Healthcare Administration Database</collection><collection>Medical Database</collection><collection>Nursing & Allied Health Premium</collection><collection>ProQuest One Academic Eastern Edition (DO NOT USE)</collection><collection>ProQuest One Academic</collection><collection>ProQuest One Academic UKI Edition</collection><collection>ProQuest Central China</collection><jtitle>Endocrine practice</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Li, Ruolin</au><au>Yang, Shumin</au><au>Gao, Rufei</au><au>Deng, Yin</au><au>Liu, Jiahuan</au><au>Yuan, Chao</au><au>Yao, Qingmei</au><au>Lv, Xinke</au><au>Wang, Kanran</au><au>Ye, Xiaoqi</au><au>Peng, Bin</au><au>Hu, Jinbo</au><au>Chen, Aijun</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>RELATIONSHIP BETWEEN THE ENVIRONMENTAL ENDOCRINE DISRUPTOR BISPHENOL A AND DYSLIPIDEMIA: A FIVE-YEAR PROSPECTIVE STUDY</atitle><jtitle>Endocrine practice</jtitle><addtitle>Endocr Pract</addtitle><date>2020-04</date><risdate>2020</risdate><volume>26</volume><issue>4</issue><spage>399</spage><epage>406</epage><pages>399-406</pages><issn>1530-891X</issn><eissn>1934-2403</eissn><abstract>To investigate whether serum bisphenol A (BPA) concentration is related to the occurrence of dyslipidemia.
A total of 574 adults were enrolled at baseline and followed up for 5 years. Concentrations of serum BPA, triglycerides (TGs), low-density lipoprotein (LDL) cholesterol, and high-density lipoprotein (HDL) cholesterol were measured. Dyslipidemia was defined as the existence of one or more of the following conditions: high-LDL-cholesterolemia (LDL ≥140 mg/dL), hypertriglyceridemia (TGs ≥150 mg/dL), or low-HDL-cholesterolemia (HDL <40 mg/dL). Participants were stratified into tertiles according to low, median, and high baseline serum BPA levels. Multivariable linear and logistic regression models were used. Data from baseline and follow-up were used for cross-sectional and longitudinal analyses, respectively.
In the cross-sectional analysis, compared to subjects in the low BPA tertile, those in the high BPA tertile showed a higher level of LDL cholesterol (108.1 ± 24.4 mg/dL versus 119.5 ± 26.9 mg/dL;
<.05) and a lower level of HDL cholesterol (46.2 ± 11.7 mg/dL versus 39.5 ± 7.5 mg/dL;
<.05). In multivariable linear regression models, Z-transformed BPA was positively associated with LDL cholesterol (β= 0.13,
= .002) and negatively associated with HDL cholesterol (β= -0.28;
<.001). After cross-sectionally adjusting for confounders, subjects in higher BPA exposure was associated with a higher prevalence of low-HDL-cholesterolemia. Longitudinally, in subjects without low-HDL-cholesterolemia at baseline, each SD increment in baseline BPA was associated with a higher incidence of low-HDL-cholesterolemia after adjustment for confounders (odds ratio [95% confidence interval; CI] 2.76, 95% CI 1.21, 6.29).
Cross-sectionally, higher BPA exposure is associated with a higher prevalence of low-HDL-cholesterolemia. Longitudinally, baseline BPA is an independent predictor of the 5-year incidence of low-HDL-cholesterolemia.
= body mass index;
= bisphenol A;
= confidence interval;
= cardiovascular disease;
= environment, inflammation and metabolic diseases study;
= high density lipoprotein;
= low density lipoprotein;
= odds ratio;
= peroxisome proliferator-activated receptor;
= systolic blood pressure;
= triglyceride;
= Z-transformed bisphenol A.</abstract><cop>United States</cop><pub>Elsevier Limited</pub><pmid>31968191</pmid><doi>10.4158/EP-2019-0384</doi><tpages>8</tpages></addata></record> |
| fulltext | fulltext |
| identifier | ISSN: 1530-891X |
| ispartof | Endocrine practice, 2020-04, Vol.26 (4), p.399-406 |
| issn | 1530-891X 1934-2403 |
| language | eng |
| recordid | cdi_proquest_journals_2389778193 |
| source | MEDLINE; ProQuest Central UK/Ireland; Alma/SFX Local Collection |
| subjects | Age Benzhydryl Compounds - adverse effects Bisphenol A Blood pressure Cardiovascular disease Cholesterol Cholesterol, HDL Chronic illnesses Cross-Sectional Studies Diabetes Dyslipidemias - chemically induced Endocrine Disruptors Exercise Family medical history Fasting Humans Hypertension Kidney diseases Lipids Low density lipoprotein Metabolic disorders Phenols - adverse effects Physical fitness Prospective Studies Risk Factors Studies Triglycerides Variables |
| title | RELATIONSHIP BETWEEN THE ENVIRONMENTAL ENDOCRINE DISRUPTOR BISPHENOL A AND DYSLIPIDEMIA: A FIVE-YEAR PROSPECTIVE STUDY |
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