Bradykinin-induced mesenteric vasodilation is mediated by B(2)-subtype receptors and nitric oxide

Mechanisms mediating bradykinin-induced anterior mesenteric artery vasodilation in anesthetized rats were examined. It was found that the mesenteric vasodilation in rats is mediated partly by B2-receptors and endogenous nitric oxide generation.

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Veröffentlicht in:American journal of physiology: Gastrointestinal and liver physiology 1993-03, Vol.27 (3), p.G492
Hauptverfasser: Berguer, Ramon, Hottenstein, Omar D, Palen, Ted E, Stewart, John M, Jacobson, Eugene D
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Sprache:eng
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