Systemic administration of amylin increases bone mass, linear growth, and adiposity in adult male mice
Amylin is a peptide hormone cosecreted with insulin from the pancreatic beta-cells that can act as an osteoblast mitogen and as an inhibitor of bone resorption. The effects on bone of its systemic administration are uncertain.
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| Veröffentlicht in: | American journal of physiology: endocrinology and metabolism 1998-10, Vol.38 (4), p.E694-E699 |
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| container_end_page | E699 |
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| container_issue | 4 |
| container_start_page | E694 |
| container_title | American journal of physiology: endocrinology and metabolism |
| container_volume | 38 |
| creator | CORNISH, J CALLON, K. E KING, A. R COOPER, G. J. S REID, I. R |
| description | Amylin is a peptide hormone cosecreted with insulin from the pancreatic beta-cells that can act as an osteoblast mitogen and as an inhibitor of bone resorption. The effects on bone of its systemic administration are uncertain. |
| format | Article |
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E ; KING, A. R ; COOPER, G. J. S ; REID, I. R</creator><creatorcontrib>CORNISH, J ; CALLON, K. E ; KING, A. R ; COOPER, G. J. S ; REID, I. R</creatorcontrib><description>Amylin is a peptide hormone cosecreted with insulin from the pancreatic beta-cells that can act as an osteoblast mitogen and as an inhibitor of bone resorption. The effects on bone of its systemic administration are uncertain.</description><identifier>ISSN: 0193-1849</identifier><identifier>EISSN: 1522-1555</identifier><identifier>CODEN: AJPMD9</identifier><language>eng</language><publisher>Bethesda, MD: American Physiological Society</publisher><subject>Biological and medical sciences ; Bones ; Bones, joints and connective tissue. Antiinflammatory agents ; Medical sciences ; Metabolism ; Pharmacology. 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Drug treatments</topic><topic>Physical growth</topic><topic>Rodents</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>CORNISH, J</creatorcontrib><creatorcontrib>CALLON, K. E</creatorcontrib><creatorcontrib>KING, A. R</creatorcontrib><creatorcontrib>COOPER, G. J. S</creatorcontrib><creatorcontrib>REID, I. R</creatorcontrib><collection>Pascal-Francis</collection><collection>Calcium & Calcified Tissue Abstracts</collection><collection>Physical Education Index</collection><collection>Toxicology Abstracts</collection><collection>Environmental Sciences and Pollution Management</collection><jtitle>American journal of physiology: endocrinology and metabolism</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>CORNISH, J</au><au>CALLON, K. E</au><au>KING, A. R</au><au>COOPER, G. J. S</au><au>REID, I. 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| fulltext | fulltext |
| identifier | ISSN: 0193-1849 |
| ispartof | American journal of physiology: endocrinology and metabolism, 1998-10, Vol.38 (4), p.E694-E699 |
| issn | 0193-1849 1522-1555 |
| language | eng |
| recordid | cdi_proquest_journals_232328232 |
| source | American Physiological Society; EZB-FREE-00999 freely available EZB journals |
| subjects | Biological and medical sciences Bones Bones, joints and connective tissue. Antiinflammatory agents Medical sciences Metabolism Pharmacology. Drug treatments Physical growth Rodents |
| title | Systemic administration of amylin increases bone mass, linear growth, and adiposity in adult male mice |
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