Reverse changes in cardiac substrate oxidation in dogs recovering from heart failure

1 Department of Physiology, New York Medical College, Valhalla, New York; 2 Department of Medicine, Division of Cardiovascular Medicine, Henry Ford Hospital, Detroit, Michigan; 3 Division of Cardiology, Department of Medicine, University of Maryland School of Medicine, Baltimore, Maryland; and 4 Sec...

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Veröffentlicht in:	American journal of physiology. Heart and circulatory physiology 2008-11, Vol.295 (5), p.H2098-H2105
Hauptverfasser:	Qanud, Khaled, Mamdani, Mohammed, Pepe, Martino, Khairallah, Ramzi J, Gravel, John, Lei, Biao, Gupte, Sachin A, Sharov, Victor G, Sabbah, Hani N, Stanley, William C, Recchia, Fabio A
Format:	Artikel
Sprache:	eng
Schlagworte:	Aconitate Hydratase - metabolism Acyl-CoA Dehydrogenase - metabolism Adrenergic beta-Agonists - pharmacology Animals Cardiac Pacing, Artificial Citrate (si)-Synthase - metabolism Disease Models, Animal Dogs Energy Metabolism Fatty acids Fatty Acids, Nonesterified - metabolism Gene expression Glucose Glucose - metabolism Heart Heart failure Heart Failure - diagnostic imaging Heart Failure - metabolism Heart Failure - physiopathology Hemodynamics - drug effects Male Mitochondria, Heart - enzymology Mitochondria, Heart - metabolism Myocardial Contraction Myocardium - enzymology Myocardium - metabolism Myocardium - pathology Oxidation Oxidation-Reduction Recovery of Function Studies Time Factors Ultrasonography Ventricular Remodeling
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