A possible role for atrial fibroblasts in postinfarction bradycardia
Atrial fibroblasts are considered to modulate the contractile activity of the heart in response to mechanical stretch. In this study we examined whether atrial fibroblasts are possibly involved in brandyarrhythmia, which is a severe complication after myocardial infarction.
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Veröffentlicht in: | American journal of physiology. Heart and circulatory physiology 2002-03, Vol.51 (3), p.842-849 |
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container_title | American journal of physiology. Heart and circulatory physiology |
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creator | KAMKIN, Andre KISELEVA, Irina WAGNER, Kay-Dietrich PYLAEV, Alexander LEITERER, Kate P THERES, Heinz SCHOLZ, Holger GÜNTHER, Joachim ISENBERG, Gerrit |
description | Atrial fibroblasts are considered to modulate the contractile activity of the heart in response to mechanical stretch. In this study we examined whether atrial fibroblasts are possibly involved in brandyarrhythmia, which is a severe complication after myocardial infarction. |
format | Article |
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identifier | ISSN: 0363-6135 |
ispartof | American journal of physiology. Heart and circulatory physiology, 2002-03, Vol.51 (3), p.842-849 |
issn | 0363-6135 1522-1539 |
language | eng |
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source | American Physiological Society; Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals |
subjects | Biological and medical sciences Cardiology Cardiology. Vascular system Coronary heart disease Heart Heart attacks Medical sciences |
title | A possible role for atrial fibroblasts in postinfarction bradycardia |
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