Role of ANG II in hypertension produced by chronic inhibition of nitric oxide synthase in conscious rats

Melaragno and Fink conducted experiments to test the hypothesis that hypertension caused by chronic inhibition of nitric oxide synthase (NOS) is associated with augmented pressor responsiveness to angiotensin II (ANG II). Results indicate that ANG II is involved in Nomega-nitro-L-arginine methyl est...

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Veröffentlicht in:American journal of physiology. Heart and circulatory physiology 1996-08, Vol.40 (2), p.H806
Hauptverfasser: Melaragno, Matthew G, Fink, Gregory D
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container_title American journal of physiology. Heart and circulatory physiology
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creator Melaragno, Matthew G
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description Melaragno and Fink conducted experiments to test the hypothesis that hypertension caused by chronic inhibition of nitric oxide synthase (NOS) is associated with augmented pressor responsiveness to angiotensin II (ANG II). Results indicate that ANG II is involved in Nomega-nitro-L-arginine methyl ester (L-NAME) hypertension, but chronic pressor responsiveness to the peptide is not augmented by L-NAME.
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ispartof American journal of physiology. Heart and circulatory physiology, 1996-08, Vol.40 (2), p.H806
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1522-1539
language eng
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source Alma/SFX Local Collection
subjects Biochemistry
Enzymes
Hypertension
Peptides
Rodents
title Role of ANG II in hypertension produced by chronic inhibition of nitric oxide synthase in conscious rats
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