1166-P: Bromocriptine-QR (BQR) Therapy Reduces Elevated Sympathetic Nervous System Activity (SNSA) and Oxidative Stress (OS) in Type 2 Diabetes (T2D) Subjects whose Dysglycemia Is Poorly Controlled on GLP-1 Receptor Agonist (GLP-1RA)

Elevated SNSA induces dysglycemia by enhancing adipose lipolysis and stimulating hepatic glucose production. It also promotes cardiovascular disease (CVD) by multiple mechanisms including augmenting systemic and vascular reactive oxygen and nitrogen species (ROS, RNS) generation. Elevated plasma pro...

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Veröffentlicht in:	Diabetes (New York, N.Y.) N.Y.), 2019-06, Vol.68 (Supplement_1)
Hauptverfasser:	CHAMARTHI, BINDU, CERSOSIMO, EUGENIO, ADAMS, JOHN M., ALATRACH, MARIAM, AGYIN, CHRISTINA, TRIPLITT, CURTIS L., EZROKHI, MICHAEL, LUO, SHUQIN, DUVALLET, EMILIE, CINCOTTA, ANTHONY H., DEFRONZO, RALPH A.
Format:	Artikel
Sprache:	eng
Schlagworte:	Agonists Bromocriptine Cardiovascular diseases Diabetes Diabetes mellitus Diabetes mellitus (non-insulin dependent) Dopamine GLP-1 receptor agonists Glucose Hyperglycemia Hyperprolactinemia Leptin Lipolysis Nitrotyrosine Norepinephrine Oxidative stress Prolactin Reactive oxygen species Sympathetic nervous system
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