Autoimmunity and cytokines in Guillain-Barré syndrome revisited: review of pathomechanisms with an eye on therapeutic options

Guillain-Barré syndrome (GBS) is the most common cause of acute paralysis in the United States. Campylobacter jejuni is a common trigger for GBS, igniting autoimmunity as a result of molecular mimicry between C. jejuni lipooligosaccharide (LOS) and host gangliosides. Evidence also suggests an active...

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Veröffentlicht in:	European Cytokine Network 2019-03, Vol.30 (1), p.1-14
Hauptverfasser:	Ebrahim Soltani, Zahra, Rahmani, Farzaneh, Rezaei, Nima
Format:	Artikel
Sprache:	eng
Schlagworte:	Animal models Antigens Autoantibodies Autoantigens Autoimmunity Campylobacter Cell-mediated immunity Demyelination Experimental allergic neuritis Gangliosides Guillain-Barre syndrome Immunoglobulins Inflammation Innate immunity Interleukin 17 Intravenous administration Lipooligosaccharides Mimicry Monoclonal antibodies Neuritis Neuropathy Paralysis Pathogens Polyneuropathy
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