VO2 kinetics and the O2 deficit in heavy exercise
Department of Nutrition, Food, and Exercise Science, The Florida State University, Tallahassee, Florida 32306 The purpose of this study was to examine a new method for calculating the O 2 deficit that considered the O 2 uptake ( O 2 ) kinetics during exercise as two separate phases in light of previ...
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| Veröffentlicht in: | Journal of applied physiology (1985) 2000-04, Vol.88 (4), p.1407-1412 |
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| container_title | Journal of applied physiology (1985) |
| container_volume | 88 |
| creator | Bearden, S. E Moffatt, R. J |
| description | Department of Nutrition, Food, and Exercise Science, The Florida
State University, Tallahassee, Florida 32306
The purpose
of this study was to examine a new method for calculating the
O 2 deficit that considered the O 2 uptake
( O 2 ) kinetics during
exercise as two separate phases in light of previous research in which
it was shown that the traditional O 2 deficit calculation
overestimated the recovery O 2 consumption (ROC). Eight subjects completed exercise transitions between unloaded cycling and
25% (heavy, H) or 50% (very heavy, VH) of the difference between the
lactic acid threshold (LAT) and peak
O 2 for 8 min. The
O 2 deficit, calculated in the traditional manner, was
significantly greater than the measured ROC for both above-LAT
exercises: 4.03 ± 1.01 vs. 2.63 ± 0.80 (SD) liters for VH
and 2.36 ± 0.91 vs. 1.74 ± 0.63 liters for H for the O 2
deficit vs. ROC ( P |
| format | Article |
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State University, Tallahassee, Florida 32306
The purpose
of this study was to examine a new method for calculating the
O 2 deficit that considered the O 2 uptake
( O 2 ) kinetics during
exercise as two separate phases in light of previous research in which
it was shown that the traditional O 2 deficit calculation
overestimated the recovery O 2 consumption (ROC). Eight subjects completed exercise transitions between unloaded cycling and
25% (heavy, H) or 50% (very heavy, VH) of the difference between the
lactic acid threshold (LAT) and peak
O 2 for 8 min. The
O 2 deficit, calculated in the traditional manner, was
significantly greater than the measured ROC for both above-LAT
exercises: 4.03 ± 1.01 vs. 2.63 ± 0.80 (SD) liters for VH
and 2.36 ± 0.91 vs. 1.74 ± 0.63 liters for H for the O 2
deficit vs. ROC ( P < 0.05). When the kinetics were viewed as
two separate components with independent onsets, the calculated
O 2 deficit (2.89 ± 0.79 and 1.71 ± 0.70 liters for VH and H, respectively) was not different from the measured
ROC ( P < 0.05). Subjects also performed the same work rate
for only 3 min. These data, from bouts terminated before the slow
component could contribute appreciably to the overall
O 2 response, show that the
O 2 requirement during the transition is less than the final
steady state for the work rate, as evidenced by symmetry between the
O 2 deficit and ROC. This new method of calculating the
O 2 deficit more closely reflects the expected
O 2 deficit-ROC relationship (i.e., ROC O 2
deficit). Therefore, estimation of the O 2 deficit during
heavy exercise transitions should consider the slow component of
O 2 as an additional deficit
component with delayed onset.
recovery oxygen consumption; lactic acid threshold; square wave; steady state</description><identifier>ISSN: 8750-7587</identifier><identifier>EISSN: 1522-1601</identifier><identifier>PMID: 10749836</identifier><identifier>CODEN: JAPHEV</identifier><language>eng</language><publisher>Bethesda, MD: Am Physiological Soc</publisher><subject>Anatomy & physiology ; Biological and medical sciences ; Exercise ; Fundamental and applied biological sciences. Psychology ; Oxygen ; Vertebrates: body movement. Posture. Locomotion. Flight. Swimming. Physical exercise. Rest. Sports</subject><ispartof>Journal of applied physiology (1985), 2000-04, Vol.88 (4), p.1407-1412</ispartof><rights>2000 INIST-CNRS</rights><rights>Copyright American Physiological Society Apr 2000</rights><lds50>peer_reviewed</lds50><woscitedreferencessubscribed>false</woscitedreferencessubscribed></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,776,780</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=1408254$$DView record in Pascal Francis$$Hfree_for_read</backlink></links><search><creatorcontrib>Bearden, S. E</creatorcontrib><creatorcontrib>Moffatt, R. J</creatorcontrib><title>VO2 kinetics and the O2 deficit in heavy exercise</title><title>Journal of applied physiology (1985)</title><description>Department of Nutrition, Food, and Exercise Science, The Florida
State University, Tallahassee, Florida 32306
The purpose
of this study was to examine a new method for calculating the
O 2 deficit that considered the O 2 uptake
( O 2 ) kinetics during
exercise as two separate phases in light of previous research in which
it was shown that the traditional O 2 deficit calculation
overestimated the recovery O 2 consumption (ROC). Eight subjects completed exercise transitions between unloaded cycling and
25% (heavy, H) or 50% (very heavy, VH) of the difference between the
lactic acid threshold (LAT) and peak
O 2 for 8 min. The
O 2 deficit, calculated in the traditional manner, was
significantly greater than the measured ROC for both above-LAT
exercises: 4.03 ± 1.01 vs. 2.63 ± 0.80 (SD) liters for VH
and 2.36 ± 0.91 vs. 1.74 ± 0.63 liters for H for the O 2
deficit vs. ROC ( P < 0.05). When the kinetics were viewed as
two separate components with independent onsets, the calculated
O 2 deficit (2.89 ± 0.79 and 1.71 ± 0.70 liters for VH and H, respectively) was not different from the measured
ROC ( P < 0.05). Subjects also performed the same work rate
for only 3 min. These data, from bouts terminated before the slow
component could contribute appreciably to the overall
O 2 response, show that the
O 2 requirement during the transition is less than the final
steady state for the work rate, as evidenced by symmetry between the
O 2 deficit and ROC. This new method of calculating the
O 2 deficit more closely reflects the expected
O 2 deficit-ROC relationship (i.e., ROC O 2
deficit). Therefore, estimation of the O 2 deficit during
heavy exercise transitions should consider the slow component of
O 2 as an additional deficit
component with delayed onset.
recovery oxygen consumption; lactic acid threshold; square wave; steady state</description><subject>Anatomy & physiology</subject><subject>Biological and medical sciences</subject><subject>Exercise</subject><subject>Fundamental and applied biological sciences. Psychology</subject><subject>Oxygen</subject><subject>Vertebrates: body movement. Posture. Locomotion. Flight. Swimming. Physical exercise. Rest. Sports</subject><issn>8750-7587</issn><issn>1522-1601</issn><fulltext>true</fulltext><rsrctype>article</rsrctype><creationdate>2000</creationdate><recordtype>article</recordtype><recordid>eNp1zk1Lw0AQBuBFFFur_2ERD14C-5ndHKVYKxR6qV6XzX40W2MSd1Nt_r0pLd6cy8DwvDNzAaaYE5LhHOFLMJWCo0xwKSbgJqUdQpgxjq_BBCPBCknzKcDvawI_QuP6YBLUjYV95eA4s84HE3oYGlg5_T1Ad3DRhORuwZXXdXJ35z4Db4vnzXyZrdYvr_OnVVZhylBGRa4LK60RPtelwSynlhKihRHSc13q3JTYUmqdKyUtvC2sx9zjgnDPqEd0Bu5Pe7vYfu1d6tWu3cdmPKnIWJhIdkQPZ6ST0bWPuhl_VF0MnzoOCjMkCWcjezyxKmyrnxCd6qohhbZut4Pa6U5JqdhRi5Gy_-liX9cbd-iPmb-I6qynv-qNbz4</recordid><startdate>20000401</startdate><enddate>20000401</enddate><creator>Bearden, S. E</creator><creator>Moffatt, R. J</creator><general>Am Physiological Soc</general><general>American Physiological Society</general><scope>IQODW</scope><scope>7QP</scope><scope>7QR</scope><scope>7TK</scope><scope>7TS</scope><scope>7U7</scope><scope>8FD</scope><scope>C1K</scope><scope>FR3</scope><scope>P64</scope></search><sort><creationdate>20000401</creationdate><title>VO2 kinetics and the O2 deficit in heavy exercise</title><author>Bearden, S. E ; Moffatt, R. J</author></sort><facets><frbrtype>5</frbrtype><frbrgroupid>cdi_FETCH-LOGICAL-h1340-376a9d8dc7f6abc1463d322a7c78f5aba6cb1d33deeb839fd9df15f1925f43f03</frbrgroupid><rsrctype>articles</rsrctype><prefilter>articles</prefilter><language>eng</language><creationdate>2000</creationdate><topic>Anatomy & physiology</topic><topic>Biological and medical sciences</topic><topic>Exercise</topic><topic>Fundamental and applied biological sciences. Psychology</topic><topic>Oxygen</topic><topic>Vertebrates: body movement. Posture. Locomotion. Flight. Swimming. Physical exercise. Rest. Sports</topic><toplevel>peer_reviewed</toplevel><toplevel>online_resources</toplevel><creatorcontrib>Bearden, S. E</creatorcontrib><creatorcontrib>Moffatt, R. J</creatorcontrib><collection>Pascal-Francis</collection><collection>Calcium & Calcified Tissue Abstracts</collection><collection>Chemoreception Abstracts</collection><collection>Neurosciences Abstracts</collection><collection>Physical Education Index</collection><collection>Toxicology Abstracts</collection><collection>Technology Research Database</collection><collection>Environmental Sciences and Pollution Management</collection><collection>Engineering Research Database</collection><collection>Biotechnology and BioEngineering Abstracts</collection><jtitle>Journal of applied physiology (1985)</jtitle></facets><delivery><delcategory>Remote Search Resource</delcategory><fulltext>fulltext</fulltext></delivery><addata><au>Bearden, S. E</au><au>Moffatt, R. J</au><format>journal</format><genre>article</genre><ristype>JOUR</ristype><atitle>VO2 kinetics and the O2 deficit in heavy exercise</atitle><jtitle>Journal of applied physiology (1985)</jtitle><date>2000-04-01</date><risdate>2000</risdate><volume>88</volume><issue>4</issue><spage>1407</spage><epage>1412</epage><pages>1407-1412</pages><issn>8750-7587</issn><eissn>1522-1601</eissn><coden>JAPHEV</coden><abstract>Department of Nutrition, Food, and Exercise Science, The Florida
State University, Tallahassee, Florida 32306
The purpose
of this study was to examine a new method for calculating the
O 2 deficit that considered the O 2 uptake
( O 2 ) kinetics during
exercise as two separate phases in light of previous research in which
it was shown that the traditional O 2 deficit calculation
overestimated the recovery O 2 consumption (ROC). Eight subjects completed exercise transitions between unloaded cycling and
25% (heavy, H) or 50% (very heavy, VH) of the difference between the
lactic acid threshold (LAT) and peak
O 2 for 8 min. The
O 2 deficit, calculated in the traditional manner, was
significantly greater than the measured ROC for both above-LAT
exercises: 4.03 ± 1.01 vs. 2.63 ± 0.80 (SD) liters for VH
and 2.36 ± 0.91 vs. 1.74 ± 0.63 liters for H for the O 2
deficit vs. ROC ( P < 0.05). When the kinetics were viewed as
two separate components with independent onsets, the calculated
O 2 deficit (2.89 ± 0.79 and 1.71 ± 0.70 liters for VH and H, respectively) was not different from the measured
ROC ( P < 0.05). Subjects also performed the same work rate
for only 3 min. These data, from bouts terminated before the slow
component could contribute appreciably to the overall
O 2 response, show that the
O 2 requirement during the transition is less than the final
steady state for the work rate, as evidenced by symmetry between the
O 2 deficit and ROC. This new method of calculating the
O 2 deficit more closely reflects the expected
O 2 deficit-ROC relationship (i.e., ROC O 2
deficit). Therefore, estimation of the O 2 deficit during
heavy exercise transitions should consider the slow component of
O 2 as an additional deficit
component with delayed onset.
recovery oxygen consumption; lactic acid threshold; square wave; steady state</abstract><cop>Bethesda, MD</cop><pub>Am Physiological Soc</pub><pmid>10749836</pmid><tpages>6</tpages></addata></record> |
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| source | American Physiological Society; EZB-FREE-00999 freely available EZB journals; Alma/SFX Local Collection |
| subjects | Anatomy & physiology Biological and medical sciences Exercise Fundamental and applied biological sciences. Psychology Oxygen Vertebrates: body movement. Posture. Locomotion. Flight. Swimming. Physical exercise. Rest. Sports |
| title | VO2 kinetics and the O2 deficit in heavy exercise |
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