Protein kinase C [beta] inhibition attenuates the progression of experimental diabetic nephropathy in the presence of continued hypertension

In addition to hyperglycemia, hypertension and the renin-angiotensin system have been consistently implicated in the pathogenesis of diabetic nephropathy. Each of these pathogenetic factors may induce changes in cellular function by a common intracellular signaling pathway, the activation of protein...

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Veröffentlicht in:	Diabetes (New York, N.Y.) N.Y.), 2003-02, Vol.52 (2), p.512
Hauptverfasser:	Kelly, Darren J, Zhang, Yuan, Hepper, Claire, Gow, Renae M, Jaworski, Kassie, Kemp, Bruce E, Wilkinson-Berka, Jennifer L, Gilbert, Richard E
Format:	Artikel
Sprache:	eng
Schlagworte:	Blood pressure Development and progression Diabetes Diabetic nephropathies Diabetic nephropathy Endocrine system Glucose Growth factors Hyperglycemia Hypertension Kidney diseases Kinases Pathogenesis Physiological aspects Protein kinases Proteins Renin-angiotensin system
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