Association of 72-kDa heat shock protein expression with adaptation to aspirin in rat gastric mucosa

It is well documented that gastric mucosa can increase its resistance to mucosal damage caused by aspirin during repeated long-term administration of aspirin. However, the underlying mechanism of this adaptation is not well established. In the present study, we investigated the effect of long-term (...

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Veröffentlicht in:	Digestive diseases and sciences 1999-07, Vol.44 (7), p.1401-1407
Hauptverfasser:	JIN, M, OTAKA, M, ITOH, H, PHD, TASHIMA, Y, MASAMUNE, O, WATANABE, S, OKUYAMA, A, ITOH, S, OTANI, S, ODASHIMA, M, IWABUCHI, A, KONISHI, N, WADA, I, PACHECO, I
Format:	Artikel
Sprache:	eng
Schlagworte:	Adaptation, Physiological - genetics Animals Anti-Inflammatory Agents, Non-Steroidal - toxicity Aspirin - toxicity Biological and medical sciences Bones, joints and connective tissue. Antiinflammatory agents Dinoprostone - metabolism Gastric Mucosa - drug effects Gastric Mucosa - pathology Gene Expression - drug effects Heat-Shock Proteins - genetics HSP72 Heat-Shock Proteins Immunoenzyme Techniques Leukotriene B4 - metabolism Long-Term Care Male Medical sciences Pharmacology. Drug treatments Rats Rats, Sprague-Dawley
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