Reversal of behavioral decline and neuropathology by a complex vitamin supplement involves modulation of key neurochemical stressors

•B-complex vitamins plus ascorbic acid reverses behavioral decline and neuropathology•Inhibition of neurochemical and cellular imbalances in key brain areas are shown•Modulation of transferrin receptor-1 is involved in therapeutic mechanisms Metal ions are crucial for normal neurochemical signaling...

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Veröffentlicht in:	Environmental toxicology and pharmacology 2018-09, Vol.62, p.120-131
Hauptverfasser:	Olajide, Olayemi Joseph, Fatoye, John Oluwasegun, Idowu, Oluwakunmi Folashade, Ilekoya, Damilola, Gbadamosi, Ismail Temitayo, Gbadamosi, Monsurat Titi, Asogwa, Nnaemeka Tobechukwu
Format:	Artikel
Sprache:	eng
Schlagworte:	Acetylcholinesterase Aluminum chloride Alzheimer’s disease Anxiety Ascorbic acid B-complex vitamins Bioenergetics Catalase Chemical vapor synthesis Cognitive ability Dehydrogenase Glucokinase Glucose bioenergetics Glucosephosphate dehydrogenase Glutathione Glutathione peroxidase Hippocampus Histochemistry Histology Homeostasis Hypertrophy L-Lactate dehydrogenase Labeling Lactate dehydrogenase Lactic acid Malondialdehyde Metal ion homeostasis Metal ions Modulation Neurodegeneration Neuromodulation Neuropathology Neurotoxicity Nitric oxide Normalizing Oxidative stress Peroxidase Prefrontal cortex Protein biosynthesis Protein synthesis Proteins Rats Rodents Short term memory Superoxide dismutase Transferrin Vitamins
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