A deficient TLR2 signaling promotes airway mucin production in Mycoplasma pneumoniae-infected allergic mice

Department of Medicine, National Jewish Medical and Research Center, Denver, Colorado Submitted 8 August 2006 ; accepted in final form 24 December 2006 The original hygiene hypothesis suggests that early childhood respiratory infections preceding allergen exposure may decrease the prevalence of alle...

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Veröffentlicht in:	American journal of physiology. Lung cellular and molecular physiology 2007-05, Vol.292 (5), p.L1064-L1072
Hauptverfasser:	Wu, Qun, Martin, Richard J, Rino, John G, Jeyaseelan, Samithamby, Breed, Rachel, Chu, Hong Wei
Format:	Artikel
Sprache:	eng
Schlagworte:	Allergens Animals Asthma Bacteria Enzyme-Linked Immunosorbent Assay Gene Expression Regulation Hypersensitivity Infections Interferon-gamma - genetics Lungs Mice Mucin 5AC Mucins - biosynthesis Mucins - genetics Mycoplasma pneumoniae Pneumonia, Mycoplasma - immunology Pneumonia, Mycoplasma - physiopathology Promoter Regions, Genetic Respiratory Mucosa - immunology Respiratory Mucosa - pathology Reverse Transcriptase Polymerase Chain Reaction Rodents Toll-Like Receptor 2 - deficiency Toll-Like Receptor 2 - genetics Toll-Like Receptor 2 - physiology Trachea
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