Appetizing rancidity of apoptotic cells for macrophages: oxidation, externalization, and recognition of phosphatidylserine
Programmed cell death (apoptosis) functions as a mechanism to eliminate unwanted or irreparably damaged cells ultimately leading to their orderly phagocytosis in the absence of calamitous inflammatory responses. Recent studies have demonstrated that the generation of free radical intermediates and s...
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Veröffentlicht in: | American journal of physiology. Lung cellular and molecular physiology 2003-07, Vol.29 (1), p.L1-L17 |
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Sprache: | eng |
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