Caffeine affects HFD-induced hepatic steatosis by multifactorial intervention

The incidence of nonalcoholic fatty liver disease (NAFLD) is considered a risk factor for hepatic fibrosis. Therefore, there is critical need to develop novel cheap and effective therapeutic approaches to prevent and reverse NAFLD. Caffeine is commonly consumed beverage and has antioxidant and anti-...

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Veröffentlicht in:	Human & experimental toxicology 2018-09, Vol.37 (9), p.983-990
Hauptverfasser:	Helal, MG, Ayoub, SE, Elkashefand, WF, Ibrahim, TM
Format:	Artikel
Sprache:	eng
Schlagworte:	Alanine Alanine transaminase Albumin Animals Anti-Inflammatory Agents - pharmacology Antioxidants Antioxidants - pharmacology Aspartate aminotransferase Bilirubin Biomarkers - blood Caffeine Caffeine - pharmacology Carnitine Carnitine palmitoyltransferase Cholesterol Cytoprotection Diet Diet, High-Fat Disease Models, Animal Fatty acids Fatty liver Fatty-acid synthase Fibrosis Gene expression Gene Expression Regulation, Enzymologic Glutathione Health risks High fat diet Inflammation Inflammatory response Injuries Lipid peroxidation Lipid Peroxidation - drug effects Lipogenesis Lipogenesis - drug effects Lipogenesis - genetics Liver Liver - drug effects Liver - metabolism Liver - pathology Liver diseases Male Malondialdehyde Non-alcoholic Fatty Liver Disease - genetics Non-alcoholic Fatty Liver Disease - metabolism Non-alcoholic Fatty Liver Disease - pathology Non-alcoholic Fatty Liver Disease - prevention & control Oxidation Oxidation-Reduction Oxidative stress Oxidative Stress - drug effects Palmitoyltransferase Peroxidation Polymerase chain reaction Proteins Rats Rats, Wistar Reverse transcription Risk analysis Risk factors Rodents Triglycerides
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