Hallmarks of cancer progression in Barrett's oesophagus

Hanahan and Weinberg proposed in 200 that carcinogenesis involves DNA changes that enable cells to:provide their own growth signals, ignore growth-inhibitory signals, avoid apoptosis, replicate without limit, sustain angiogenesis, and invade and proliferate in unnatural locations. The metaplastic ce...

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Veröffentlicht in:	The Lancet (British edition) 2002-11, Vol.360 (9345), p.1587-1589
Hauptverfasser:	Morales, Carmela P, Souza, Rhonda F, Spechler, Stuart J
Format:	Artikel
Sprache:	eng
Schlagworte:	Abnormalities Adhesion Aneuploidy Apoptosis - physiology Barrett Esophagus - pathology Barrett's esophagus Biological and medical sciences Cancer Carcinogenesis Carcinogens Cell adhesion Cell adhesion & migration Cell Cycle Cell Division - physiology Cell Transformation, Neoplastic Cyclin-dependent kinases Cytoplasm E-cadherin Endothelial cells Esophageal Neoplasms - pathology Esophagus Gastroenterology. Liver. Pancreas. Abdomen Genetic abnormalities Genetics Growth factors Humans Kinases Medical sciences Metaplasia Mucous membrane Mutation Neoplasm Invasiveness Neoplasm Metastasis Neovascularization, Pathologic Receptors Signal Transduction - physiology Throat Tumors
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