Potentiated endothelium-derived hyperpolarizing factor-mediated dilations in cerebral arteries following mild head injury

Evidence in the literature suggests that endothelium-derived hyperpolarizing factor (EDHF) may act in a compensatory manner such that during conditions of compromised nitric oxide (NO), EDHF serves as a back-up mechanism. Given that constitutive NO synthase is chronically downregulated after head tr...

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Veröffentlicht in:	Journal of neurotrauma 2001-07, Vol.18 (7), p.691-697
Hauptverfasser:	GOLDING, Elke M, JUNPING YOU, ROBERTSON, Claudia S, BRYAN, Robert M
Format:	Artikel
Sprache:	eng
Schlagworte:	Adenosine triphosphate Adenosine Triphosphate - metabolism Analysis of Variance Animals Biological and medical sciences Biological Factors - metabolism Brain Injuries - metabolism Brain Injuries - pathology Cardiovascular disease Charybdotoxin - metabolism Dilatation, Pathologic - metabolism Dilatation, Pathologic - pathology Disease Models, Animal Endothelium Endothelium, Vascular - metabolism Head injuries Hypotheses Indomethacin - metabolism Injuries of the nervous system and the skull. Diseases due to physical agents Male Medical sciences Middle Cerebral Artery - metabolism Middle Cerebral Artery - pathology NG-Nitroarginine Methyl Ester - metabolism Nitric oxide Rats Rats, Long-Evans Smooth muscle Time Factors Trauma Traumas. Diseases due to physical agents Traumatic brain injury Veins & arteries
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