ZBTB16 and metabolic syndrome: a network perspective
Metabolic syndrome is a prevalent, complex condition. The search for genetic determinants of the syndrome is currently undergoing a paradigm enhancement by adding systems genetics approaches to association studies. We summarize the current evidence on relations between an emergent new candidate, zin...
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Veröffentlicht in: | Physiological research 2017-01, Vol.66 (Suppl 3), p.S357-S365 |
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creator | Šeda, O Šedová, L Včelák, J Vaňková, M Liška, F Bendlová, B |
description | Metabolic syndrome is a prevalent, complex condition. The search for genetic determinants of the syndrome is currently undergoing a paradigm enhancement by adding systems genetics approaches to association studies. We summarize the current evidence on relations between an emergent new candidate, zinc finger and BTB domain containing 16 (ZBTB16) transcription factor and the major components constituting the metabolic syndrome. Information stemming from studies on experimental models with altered Zbtb16 expression clearly shows its effect on adipogenesis, cardiac hypertrophy and fibrosis, lipid levels and insulin sensitivity. Based on current evidence, we provide a network view of relations between ZBTB16 and hallmarks of metabolic syndrome in order to elucidate the potential functional links involving the ZBTB16 node. Many of the identified genes interconnecting ZBTB16 with all or most metabolic syndrome components are linked to immune function, inflammation or oxidative stress. In summary, ZBTB16 represents a promising pleiotropic candidate node for metabolic syndrome. |
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The search for genetic determinants of the syndrome is currently undergoing a paradigm enhancement by adding systems genetics approaches to association studies. We summarize the current evidence on relations between an emergent new candidate, zinc finger and BTB domain containing 16 (ZBTB16) transcription factor and the major components constituting the metabolic syndrome. Information stemming from studies on experimental models with altered Zbtb16 expression clearly shows its effect on adipogenesis, cardiac hypertrophy and fibrosis, lipid levels and insulin sensitivity. Based on current evidence, we provide a network view of relations between ZBTB16 and hallmarks of metabolic syndrome in order to elucidate the potential functional links involving the ZBTB16 node. Many of the identified genes interconnecting ZBTB16 with all or most metabolic syndrome components are linked to immune function, inflammation or oxidative stress. In summary, ZBTB16 represents a promising pleiotropic candidate node for metabolic syndrome.</description><identifier>ISSN: 0862-8408</identifier><identifier>EISSN: 1802-9973</identifier><identifier>DOI: 10.33549/physiolres.933730</identifier><identifier>PMID: 28948820</identifier><language>eng</language><publisher>Czech Republic: Institute of Physiology</publisher><subject>Adipogenesis ; Animals ; DNA methylation ; Fibrosis ; Gene Regulatory Networks - physiology ; Genomics ; Heart diseases ; Humans ; Hypertrophy ; Immune response ; Insulin ; Insulin resistance ; Insulin Resistance - physiology ; Metabolic syndrome ; Metabolic Syndrome - genetics ; Metabolic Syndrome - metabolism ; Oxidative stress ; Oxidative Stress - physiology ; Promyelocytic Leukemia Zinc Finger Protein - genetics ; Promyelocytic Leukemia Zinc Finger Protein - metabolism ; Rodents ; Zinc finger proteins ; Zinc Fingers - physiology</subject><ispartof>Physiological research, 2017-01, Vol.66 (Suppl 3), p.S357-S365</ispartof><rights>Copyright Institute of Physiology 2017</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed><citedby>FETCH-LOGICAL-c408t-59884946b2f6471aec01ffaa0440f77cf50f87eb64d4401ee3af0fffa0ae1d3e3</citedby><cites>FETCH-LOGICAL-c408t-59884946b2f6471aec01ffaa0440f77cf50f87eb64d4401ee3af0fffa0ae1d3e3</cites></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><link.rule.ids>314,776,780,860,27901,27902</link.rule.ids><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/28948820$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>Šeda, O</creatorcontrib><creatorcontrib>Šedová, L</creatorcontrib><creatorcontrib>Včelák, J</creatorcontrib><creatorcontrib>Vaňková, M</creatorcontrib><creatorcontrib>Liška, F</creatorcontrib><creatorcontrib>Bendlová, B</creatorcontrib><title>ZBTB16 and metabolic syndrome: a network perspective</title><title>Physiological research</title><addtitle>Physiol Res</addtitle><description>Metabolic syndrome is a prevalent, complex condition. The search for genetic determinants of the syndrome is currently undergoing a paradigm enhancement by adding systems genetics approaches to association studies. We summarize the current evidence on relations between an emergent new candidate, zinc finger and BTB domain containing 16 (ZBTB16) transcription factor and the major components constituting the metabolic syndrome. Information stemming from studies on experimental models with altered Zbtb16 expression clearly shows its effect on adipogenesis, cardiac hypertrophy and fibrosis, lipid levels and insulin sensitivity. Based on current evidence, we provide a network view of relations between ZBTB16 and hallmarks of metabolic syndrome in order to elucidate the potential functional links involving the ZBTB16 node. Many of the identified genes interconnecting ZBTB16 with all or most metabolic syndrome components are linked to immune function, inflammation or oxidative stress. 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The search for genetic determinants of the syndrome is currently undergoing a paradigm enhancement by adding systems genetics approaches to association studies. We summarize the current evidence on relations between an emergent new candidate, zinc finger and BTB domain containing 16 (ZBTB16) transcription factor and the major components constituting the metabolic syndrome. Information stemming from studies on experimental models with altered Zbtb16 expression clearly shows its effect on adipogenesis, cardiac hypertrophy and fibrosis, lipid levels and insulin sensitivity. Based on current evidence, we provide a network view of relations between ZBTB16 and hallmarks of metabolic syndrome in order to elucidate the potential functional links involving the ZBTB16 node. Many of the identified genes interconnecting ZBTB16 with all or most metabolic syndrome components are linked to immune function, inflammation or oxidative stress. In summary, ZBTB16 represents a promising pleiotropic candidate node for metabolic syndrome.</abstract><cop>Czech Republic</cop><pub>Institute of Physiology</pub><pmid>28948820</pmid><doi>10.33549/physiolres.933730</doi><oa>free_for_read</oa></addata></record> |
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subjects | Adipogenesis Animals DNA methylation Fibrosis Gene Regulatory Networks - physiology Genomics Heart diseases Humans Hypertrophy Immune response Insulin Insulin resistance Insulin Resistance - physiology Metabolic syndrome Metabolic Syndrome - genetics Metabolic Syndrome - metabolism Oxidative stress Oxidative Stress - physiology Promyelocytic Leukemia Zinc Finger Protein - genetics Promyelocytic Leukemia Zinc Finger Protein - metabolism Rodents Zinc finger proteins Zinc Fingers - physiology |
title | ZBTB16 and metabolic syndrome: a network perspective |
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