ZBTB16 and metabolic syndrome: a network perspective

Metabolic syndrome is a prevalent, complex condition. The search for genetic determinants of the syndrome is currently undergoing a paradigm enhancement by adding systems genetics approaches to association studies. We summarize the current evidence on relations between an emergent new candidate, zin...

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Veröffentlicht in:Physiological research 2017-01, Vol.66 (Suppl 3), p.S357-S365
Hauptverfasser: Šeda, O, Šedová, L, Včelák, J, Vaňková, M, Liška, F, Bendlová, B
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container_end_page S365
container_issue Suppl 3
container_start_page S357
container_title Physiological research
container_volume 66
creator Šeda, O
Šedová, L
Včelák, J
Vaňková, M
Liška, F
Bendlová, B
description Metabolic syndrome is a prevalent, complex condition. The search for genetic determinants of the syndrome is currently undergoing a paradigm enhancement by adding systems genetics approaches to association studies. We summarize the current evidence on relations between an emergent new candidate, zinc finger and BTB domain containing 16 (ZBTB16) transcription factor and the major components constituting the metabolic syndrome. Information stemming from studies on experimental models with altered Zbtb16 expression clearly shows its effect on adipogenesis, cardiac hypertrophy and fibrosis, lipid levels and insulin sensitivity. Based on current evidence, we provide a network view of relations between ZBTB16 and hallmarks of metabolic syndrome in order to elucidate the potential functional links involving the ZBTB16 node. Many of the identified genes interconnecting ZBTB16 with all or most metabolic syndrome components are linked to immune function, inflammation or oxidative stress. In summary, ZBTB16 represents a promising pleiotropic candidate node for metabolic syndrome.
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The search for genetic determinants of the syndrome is currently undergoing a paradigm enhancement by adding systems genetics approaches to association studies. We summarize the current evidence on relations between an emergent new candidate, zinc finger and BTB domain containing 16 (ZBTB16) transcription factor and the major components constituting the metabolic syndrome. Information stemming from studies on experimental models with altered Zbtb16 expression clearly shows its effect on adipogenesis, cardiac hypertrophy and fibrosis, lipid levels and insulin sensitivity. Based on current evidence, we provide a network view of relations between ZBTB16 and hallmarks of metabolic syndrome in order to elucidate the potential functional links involving the ZBTB16 node. Many of the identified genes interconnecting ZBTB16 with all or most metabolic syndrome components are linked to immune function, inflammation or oxidative stress. 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subjects Adipogenesis
Animals
DNA methylation
Fibrosis
Gene Regulatory Networks - physiology
Genomics
Heart diseases
Humans
Hypertrophy
Immune response
Insulin
Insulin resistance
Insulin Resistance - physiology
Metabolic syndrome
Metabolic Syndrome - genetics
Metabolic Syndrome - metabolism
Oxidative stress
Oxidative Stress - physiology
Promyelocytic Leukemia Zinc Finger Protein - genetics
Promyelocytic Leukemia Zinc Finger Protein - metabolism
Rodents
Zinc finger proteins
Zinc Fingers - physiology
title ZBTB16 and metabolic syndrome: a network perspective
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