Casein kinase 1‐epsilon deletion increases mu opioid receptor‐dependent behaviors and binge eating1

Genetic and pharmacological studies indicate that casein kinase 1 epsilon (Csnk1e) contributes to psychostimulant, opioid, and ethanol motivated behaviors. We previously used pharmacological inhibition to demonstrate that Csnk1e negatively regulates the locomotor stimulant properties of opioids and...

Ausführliche Beschreibung

Gespeichert in:

Bibliographische Detailangaben
Veröffentlicht in:	Genes, brain and behavior brain and behavior, 2017-09, Vol.16 (7), p.725-738
Hauptverfasser:	Goldberg, L. R., Kirkpatrick, S. L., Yazdani, N., Luttik, K. P., Lacki, O. A., Keith Babbs, R., Jenkins, D. F., Evan Johnson, W., Bryant, C. D.
Format:	Artikel
Sprache:	eng
Schlagworte:	Adaptation Addiction Analgesia Animals Aversion Axon guidance binge eating Bulimia - genetics candidate gene Casein casein kinase Casein Kinase 1 epsilon - genetics Casein Kinase 1 epsilon - metabolism Casein kinase I CK‐1 Conditioning, Classical Corpus Striatum - metabolism CPP Eating disorders Ethanol Female Fentanyl Food Gene Deletion Gene expression Locomotor activity Male Methamphetamine Mice Mice, Inbred C57BL Myelination Naloxone Narcotics Neostriatum Opioid receptors (type mu) Opioid-Related Disorders - genetics Pain perception pavlovian conditioning Place preference conditioning QTL Receptor mechanisms Receptors, Opioid, mu - metabolism Reinforcement Reward Rodents sex differences substance use disorder Transcriptome Wnt protein
Online-Zugang:	Volltext bestellen
Tags:	Tag hinzufügen Keine Tags, Fügen Sie den ersten Tag hinzu!

Schreiben Sie den ersten Kommentar!