Nasal Group 2 Innate Lymphoid Cells Accumulate during COX-1 Inhibition in Aspirin- Exacerbated Respiratory Disease

Group 2 innate lymphoid cells (ILC2s), which promote tissue eosinophilia and mast cell responses, undergo chemotaxis and cytokine production in response to PGD2 but it is unknown whether ILC2s are active in AERD.

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Veröffentlicht in:Journal of allergy and clinical immunology 2017-02, Vol.139 (2), p.AB194-AB194
Hauptverfasser: Eastman, Jacqueline J., MD, Cavagnero, Kellen J., B.S, Deconde, Adam S., MD, Kim, Alexander S., MD, Broide, David H., MB, ChB, FAAAAI, Zuraw, Bruce L., MD, White, Andrew A., MD, FAAAAI, Christiansen, Sandra C., MD, FAAAAI, Doherty, Taylor Alan, MD, FAAAAI
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container_end_page AB194
container_issue 2
container_start_page AB194
container_title Journal of allergy and clinical immunology
container_volume 139
creator Eastman, Jacqueline J., MD
Cavagnero, Kellen J., B.S
Deconde, Adam S., MD
Kim, Alexander S., MD
Broide, David H., MB, ChB, FAAAAI
Zuraw, Bruce L., MD
White, Andrew A., MD, FAAAAI
Christiansen, Sandra C., MD, FAAAAI
Doherty, Taylor Alan, MD, FAAAAI
description Group 2 innate lymphoid cells (ILC2s), which promote tissue eosinophilia and mast cell responses, undergo chemotaxis and cytokine production in response to PGD2 but it is unknown whether ILC2s are active in AERD.
doi_str_mv 10.1016/j.jaci.2016.12.631
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source ScienceDirect Journals (5 years ago - present); EZB-FREE-00999 freely available EZB journals
subjects Allergy and Immunology
Respiratory diseases
T cell receptors
title Nasal Group 2 Innate Lymphoid Cells Accumulate during COX-1 Inhibition in Aspirin- Exacerbated Respiratory Disease
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