Effect of retinoic acid in experimental diabetic nephropathy

Although the pathogenetic mechanism of diabetic nephropathy has not been elucidated, an inflammatory mechanism has been suggested to contribute to its progression. Monocyte chemoattractant peptide (MCP)‐1 attracts macrophages and T cells, and ultimately injures renal tissue. In early diabetic nephro...

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Veröffentlicht in:	Immunology and cell biology 2004-12, Vol.82 (6), p.568-576
Hauptverfasser:	Han, Sang‐Youb, So, Gyeong‐A, Jee, Yi‐Hwa, Han, Kum‐Hyun, Kang, Young‐Sun, Kim, Hyoung‐Kyu, Kang, Shin‐Wook, Han, Dae‐Suk, Han, Jee‐Young, Cha, Dae‐Ryong
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Cell Line Chemokine CCL2 - genetics Chemokine CCL2 - metabolism Chemokine CCL2 - secretion Creatine - urine Diabetes Mellitus, Experimental - chemically induced Diabetes Mellitus, Experimental - metabolism Diabetes Mellitus, Experimental - pathology Diabetic Nephropathies - metabolism Diabetic Nephropathies - pathology diabetic nephropathy Glucose - pharmacology Immunohistochemistry inflammation Male Mice monocyte chemoattractant peptide (MCP)‐1 podocyte Proteinuria - metabolism Rats Rats, Sprague-Dawley retinoid RNA, Messenger - genetics RNA, Messenger - metabolism Streptozocin - pharmacology streptozotocin Tretinoin - pharmacology
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