Induction of the Tat-Binding Protein 1 Gene Accompanies the Disabling of Oncogenic erbB Receptor Tyrosine Kinases

Conversion of a malignant phenotype into a more normal one can be accomplished either by down-regulation of erbB family surface receptors or by creating inactive erbB heterodimers on the cell surface. In this report, we report the identification and cloning of differentially expressed genes from ant...

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Veröffentlicht in:	Proceedings of the National Academy of Sciences - PNAS 1999-05, Vol.96 (11), p.6434-6438
Hauptverfasser:	Park, Byeong-Woo, O'Rourke, Donald M., Wang, Qiang, Davis, James G., Post, Andrew, Qian, Xiaolan, Greene, Mark I.
Format:	Artikel
Sprache:	eng
Schlagworte:	3T3 Cells Animals ATPases Associated with Diverse Cellular Activities Biological Sciences Brain Neoplasms - genetics Brain Neoplasms - metabolism Brain Neoplasms - pathology Breast Neoplasms Cell Division Cell growth Cell Line, Transformed Cell lines Cell Transformation, Neoplastic Cells Complementary DNA Deoxyribonucleic acid DNA DNA-Binding Proteins - genetics DNA-Binding Proteins - metabolism Female Genes Genes, erbB-2 Genes, Tumor Suppressor Humans Medical research Messenger RNA Mice Mice, Nude Phenotypes Proteasome Endopeptidase Complex Proteins Receptor Protein-Tyrosine Kinases - genetics Receptor Protein-Tyrosine Kinases - metabolism Receptor, ErbB-2 - genetics Receptor, ErbB-2 - metabolism Receptors Reverse Transcriptase Polymerase Chain Reaction Transfection Transformed cell line Transplantation, Heterologous Tumor cell line Tumor Cells, Cultured Tumors
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