COVID-19 induces a hyperactive phenotype in circulating platelets

Coronavirus Disease 2019 (COVID-19), caused by the novel Severe Acute Respiratory Syndrome Coronavirus 2 (SARS-CoV-2), has affected over 30 million globally to date. Although high rates of venous thromboembolism and evidence of COVID-19-induced endothelial dysfunction have been reported, the precise...

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Veröffentlicht in:	PLoS biology 2021-02, Vol.19 (2), p.e3001109-e3001109
Hauptverfasser:	Comer, Shane P, Cullivan, Sarah, Szklanna, Paulina B, Weiss, Luisa, Cullen, Steven, Kelliher, Sarah, Smolenski, Albert, Murphy, Claire, Altaie, Haidar, Curran, John, O'Reilly, Katherine, Cotter, Aoife G, Marsh, Brian, Gaine, Sean, Mallon, Patrick, McCullagh, Brian, Moran, Niamh, Ní Áinle, Fionnuala, Kevane, Barry, Maguire, Patricia B
Format:	Artikel
Sprache:	eng
Schlagworte:	Activation Adenosine Triphosphate - metabolism Aged Autopsy Biology and Life Sciences Blood Coagulation Blood platelets Blood Platelets - cytology Blood Platelets - virology Body mass index Cardiovascular diseases Circulating anticoagulants Contraindications Coronaviruses COVID-19 COVID-19 - blood Critical care Dengue Dengue fever Diabetes mellitus Enzyme-Linked Immunosorbent Assay Female Gene expression Health aspects Health risks Hemostasis Hospitals Humans Hyperactivity Hypertension Immune response Infections Inflammation Intensive care Intensive Care Units Male Mean Platelet Volume Medical laboratories Medicine and Health Sciences Middle Aged Molecular weight Mortality Neutrophils Organs P-Selectin - blood Patients Phenotype Phenotypes Physiological aspects Plasma Platelet Factor 4 - blood Platelet Function Tests Platelet-rich plasma therapy Platelets Risk analysis Risk factors Severe acute respiratory syndrome coronavirus 2 Testing Thrombocytopenia Thromboembolism Thrombopoietin - blood Thrombosis Vector-borne diseases Viral diseases Viral infections
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