Increasing myosin light chain 3f (MLC3f) protects against a decline in contractile velocity

Disuse induces adaptations in skeletal muscle, which lead to muscle deterioration. Hindlimb-unloading (HU) is a well-established model to investigate cellular mechanisms responsible for disuse-induced skeletal muscle dysfunction. In myosin heavy chain (MHC) type IIB fibers HU induces a reduction in...

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Veröffentlicht in:	PloS one 2019-04, Vol.14 (4), p.e0214982-e0214982
Hauptverfasser:	Kim, Jong-Hee, Graber, Ted G, Liu, Haiming, Asakura, Atsushi, Thompson, LaDora V
Format:	Artikel
Sprache:	eng
Schlagworte:	Adaptation Adaptation, Physiological Adenoviridae Adenoviruses Animals Atrophy Biology and Life Sciences Chains Cloning Contractility Contraction Deoxyribonucleic acid DNA EDTA Fibers Genetic Vectors Hindlimb Suspension Injection Light Male Medicine and Health Sciences Muscle Contraction Muscle Fibers, Skeletal - metabolism Muscle Fibers, Skeletal - pathology Muscle proteins Muscles Muscular Disorders, Atrophic - genetics Muscular Disorders, Atrophic - metabolism Muscular Disorders, Atrophic - prevention & control Musculoskeletal system Myosin Myosin Light Chains - biosynthesis Myosin Light Chains - genetics Phosphorylation Proteins Rats Rats, Inbred F344 Reduction Skeletal muscle Transduction, Genetic Unloading Velocity
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