Heterologous expression of anti-apoptotic human 14-3-3β/α enhances iron-mediated programmed cell death in yeast

The induction of Programmed Cell Death (PCD) requires the activation of complex responses involving the interplay of a variety of different cellular proteins, pathways, and processes. Uncovering the mechanisms regulating PCD requires an understanding of the different processes that both positively a...

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Veröffentlicht in:	PloS one 2017-08, Vol.12 (8), p.e0184151-e0184151
Hauptverfasser:	Eid, Rawan, Zhou, David R, Arab, Nagla T T, Boucher, Eric, Young, Paul G, Mandato, Craig A, Greenwood, Michael T
Format:	Artikel
Sprache:	eng
Schlagworte:	14-3-3 protein 14-3-3 Proteins - genetics 14-3-3 Proteins - metabolism Alpha iron Apoptosis Autophagy Biology Biology and Life Sciences Cell death Cell survival Chemical engineering Chemistry Copper Copper - metabolism Copper - toxicity Ferritin Ferritins - genetics Ferritins - metabolism Gangrene Gene Expression Humans Hydroxyurea Iron Iron - metabolism Iron - toxicity Ischemia Lethal levels Mitochondria Mortality Mutation Pathways Physical Sciences Proteins Rapamycin Regulators Research and Analysis Methods Rodents Saccharomyces cerevisiae - cytology Saccharomyces cerevisiae - genetics Saccharomyces cerevisiae - metabolism Stresses Survival Synergistic effect Synergistic effects Tumor necrosis factor-TNF Yeast Zinc
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