Trbp Is Required for Differentiation of Myoblasts and Normal Regeneration of Skeletal Muscle
Global inactivation of Trbp, a regulator of miRNA pathways, resulted in developmental defects and postnatal lethality in mice. Recently, we showed that cardiac-specific deletion of Trbp caused heart failure. However, its functional role(s) in skeletal muscle has not been characterized. Using a condi...
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Veröffentlicht in: | PloS one 2016-05, Vol.11 (5), p.e0155349 |
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Sprache: | eng |
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