The host protein calprotectin modulates the Helicobacter pylori cag type IV secretion system via zinc sequestration

Transition metals are necessary for all forms of life including microorganisms, evidenced by the fact that 30% of all proteins are predicted to interact with a metal cofactor. Through a process termed nutritional immunity, the host actively sequesters essential nutrient metals away from invading pat...

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Veröffentlicht in:	PLoS pathogens 2014-10, Vol.10 (10), p.e1004450-e1004450
Hauptverfasser:	Gaddy, Jennifer A, Radin, Jana N, Loh, John T, Piazuelo, M Blanca, Kehl-Fie, Thomas E, Delgado, Alberto G, Ilca, Florin T, Peek, Richard M, Cover, Timothy L, Chazin, Walter J, Skaar, Eric P, Scott Algood, Holly M
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Bacterial Proteins - metabolism Biology and Life Sciences Coculture Techniques - methods Disease Models, Animal Epithelial Cells - metabolism Gastric Mucosa - metabolism Health aspects Helicobacter Infections - metabolism Helicobacter pylori Homeostasis - physiology Host-parasite relationships Leukocyte L1 Antigen Complex - metabolism Medical research Medicine and Health Sciences Mice Microbiological research Research and Analysis Methods Risk Factors Rodents Stomach Neoplasms - metabolism Ulcers Zinc Zinc - metabolism Zinc in the body
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