Distinct lipid a moieties contribute to pathogen-induced site-specific vascular inflammation

Several successful pathogens have evolved mechanisms to evade host defense, resulting in the establishment of persistent and chronic infections. One such pathogen, Porphyromonas gingivalis, induces chronic low-grade inflammation associated with local inflammatory bone loss and systemic inflammation...

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Veröffentlicht in:	PLoS pathogens 2014-07, Vol.10 (7), p.e1004215-e1004215
Hauptverfasser:	Slocum, Connie, Coats, Stephen R, Hua, Ning, Kramer, Carolyn, Papadopoulos, George, Weinberg, Ellen O, Gudino, Cynthia V, Hamilton, James A, Darveau, Richard P, Genco, Caroline A
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Apolipoproteins E - genetics Apolipoproteins E - immunology Atherosclerosis Atherosclerosis - genetics Atherosclerosis - immunology Atherosclerosis - microbiology Atherosclerosis - pathology Bacteriology Bacteroidaceae Infections - genetics Bacteroidaceae Infections - immunology Bacteroidaceae Infections - microbiology Bacteroidaceae Infections - pathology Biology and Life Sciences Development and progression Disease Models, Animal Drug therapy E coli Experiments Health aspects HEK293 Cells Humans Infections Inflammation Lipid A - immunology Lipids Macrophages - immunology Macrophages - microbiology Macrophages - pathology Mice Microscopy Osteoporosis - genetics Osteoporosis - immunology Osteoporosis - microbiology Osteoporosis - pathology Porphyromonas gingivalis - immunology Risk factors Rodents Toll-Like Receptor 4 - genetics Toll-Like Receptor 4 - immunology Vasculitis - genetics Vasculitis - immunology Vasculitis - microbiology Vasculitis - pathology
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