Regulation of neuronal cav3.1 channels by cyclin-dependent kinase 5 (Cdk5)

Low voltage-activated (LVA) T-type Ca2+ channels activate in response to subthreshold membrane depolarizations and therefore represent an important source of Ca2+ influx near the resting membrane potential. In neurons, these proteins significantly contribute to control relevant physiological process...

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Veröffentlicht in:	PloS one 2015-03, Vol.10 (3), p.e0119134
Hauptverfasser:	Calderón-Rivera, Aida, Sandoval, Alejandro, González-Ramírez, Ricardo, González-Billault, Christian, Felix, Ricardo
Format:	Artikel
Sprache:	eng
Schlagworte:	Alzheimer's disease Alzheimers disease Animals Axons - physiology Biology Calcium - metabolism Calcium channels Calcium channels (T-type) Calcium channels (voltage-gated) Calcium Channels, T-Type - chemistry Calcium Channels, T-Type - genetics Calcium Channels, T-Type - metabolism Calcium influx Cell Differentiation Cell Line, Tumor Cell migration Cell Movement Channels Cortex Cyclin-dependent kinase 5 Cyclin-Dependent Kinase 5 - genetics Cyclin-Dependent Kinase 5 - metabolism Cyclin-dependent kinases Depolarization Excitability Gene expression HEK293 Cells Humans Kinases Lamination Low voltage Membrane potential Mice Mutagenesis Mutagenesis, Site-Directed Neuroblastoma Neuroblastoma cells Neurons - cytology Neurons - physiology Penicillin Phosphorylation Physiology Proteins Regulations Serine Serine - metabolism siRNA Site-directed mutagenesis Studies
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