Regulatory circuits that enable proliferation of the fungus Candida albicans in a mammalian host

The ability of C. albicans to colonize and proliferate in the GI tract has been studied in mice receiving antibiotics orally; while these animals are not natural hosts of C. albicans, they likely serve as reasonable proxies. Because transcription regulators are central elements within the gene net...

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Veröffentlicht in:	PLoS pathogens 2013-12, Vol.9 (12), p.e1003780-e1003780
Hauptverfasser:	Pérez, J Christian, Johnson, Alexander D
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biomedical research Candida Candida albicans - genetics Candida albicans - growth & development Candida albicans - pathogenicity Candidiasis - microbiology Cell Proliferation Disease Fungi Gene expression Gene Regulatory Networks - physiology Genomes Health aspects Host-parasite relationships Host-Pathogen Interactions - genetics Host-Pathogen Interactions - immunology Human body Humans Infections Intestines - metabolism Intestines - microbiology Iron - metabolism Microbiological research Microbiota Microorganisms Pearls Physiological aspects Studies
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