Characterization of neuronal populations in the human trigeminal ganglion and their association with latent herpes simplex virus-1 infection

Following primary infection Herpes simplex virus-1 (HSV-1) establishes lifelong latency in the neurons of human sensory ganglia. Upon reactivation HSV-1 can cause neurological diseases such as facial palsy, vestibular neuritis or encephalitis. Certain populations of sensory neurons have been shown t...

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Veröffentlicht in:	PloS one 2013-12, Vol.8 (12), p.e83603-e83603
Hauptverfasser:	Flowerdew, Sarah E, Wick, Desiree, Himmelein, Susanne, Horn, Anja K E, Sinicina, Inga, Strupp, Michael, Brandt, Thomas, Theil, Diethilde, Hüfner, Katharina
Format:	Artikel
Sprache:	eng
Schlagworte:	Activation Adult Aged Animals Antibodies Biomarkers Biomarkers - metabolism Calcitonin Calcitonin gene-related peptide Cell Size Deoxyribonucleic acid Diseases DNA Encephalitis Ethics Female Ganglia Ganglion cysts Gene Expression Regulation Glial cell line-derived neurotrophic factor Growth factors Health aspects Herpes simplex Herpes simplex virus Herpes viruses Herpesvirus 1, Human - metabolism Herpesvirus 1, Human - physiology Humans Immunofluorescence Infections Intermediate filament proteins Latency Latent infection Legal medicine Localization Male Middle Aged Nerve growth factor Neuritis Neurological diseases Neurology Neurons Neurons - cytology Neurons - virology Nitric oxide Nitric-oxide synthase Pain Paralysis Peripherin Populations Protein Transport Proteins Risk factors Sensory neurons Signaling Trigeminal ganglion Trigeminal Ganglion - cytology TrkA protein TrkA receptors Vestibular system Viral Proteins - genetics Viral Proteins - metabolism Viruses Young Adult
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