Paranode Abnormalities and Oxidative Stress in Optic Nerve Vulnerable to Secondary Degeneration: Modulation by 670 nm Light Treatment

Secondary degeneration of nerve tissue adjacent to a traumatic injury results in further loss of neurons, glia and function, via mechanisms that may involve oxidative stress. However, changes in indicators of oxidative stress have not yet been demonstrated in oligodendrocytes vulnerable to secondary...

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Veröffentlicht in:	PloS one 2013-06, Vol.8 (6), p.e66448-e66448
Hauptverfasser:	Szymanski, Charis R, Chiha, Wissam, Morellini, Natalie, Cummins, Nadia, Bartlett, Carole A, O'Hare Doig, Ryan L, Savigni, Donna L, Payne, Sophie C, Harvey, Alan R, Dunlop, Sarah A, Fitzgerald, Melinda
Format:	Artikel
Sprache:	eng
Schlagworte:	Abnormalities Animals Apoptosis Biology Cytochrome Cytochrome c Cytochrome oxidase Cytochrome-c oxidase Degeneration Disease Models, Animal Electron Transport Complex IV - metabolism Female Injury prevention Light Lipid peroxidation Lysine Medicine Metabolism Nerve Degeneration - metabolism Nerve Degeneration - therapy Neuronal-glial interactions Neurons Neurosciences Nystagmus Oligodendrocytes Oligodendroglia - metabolism Optic nerve Optic Nerve Injuries - complications Optic Nerve Injuries - metabolism Optic Nerve Injuries - therapy Optokinetic response Oxidase Oxidative metabolism Oxidative Stress Phototherapy - methods Physiological aspects Rats Retina Retinal ganglion cells Retinal Ganglion Cells - metabolism Rodents Superoxide Trauma Up-Regulation Visual perception
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