Effects of HCV on basal and tat-induced HIV LTR activation

Hepatitis C virus (HCV) co-infection occurs in ∼30-40% of the HIV-infected population in the US. While a significant body of research suggests an adverse effect of HIV on HCV replication and disease progression, the impact of HCV on HIV infection has not been well studied. Increasing data suggest th...

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Veröffentlicht in:	PloS one 2013-06, Vol.8 (6), p.e64956-e64956
Hauptverfasser:	Sengupta, Satarupa, Powell, Eleanor, Kong, Ling, Blackard, Jason T
Format:	Artikel
Sprache:	eng
Schlagworte:	Analysis Binding Sites Biology Blotting, Western Cells, Cultured Clonal deletion Coinfection Core protein Cytokines Development and progression Disease Gene expression Gene Expression Regulation, Viral Genes Health aspects Hepacivirus - pathogenicity Hepatitis Hepatitis C Hepatitis C - metabolism Hepatitis C - pathology Hepatitis C - virology Hepatitis C virus Hepatocytes Hepatocytes - cytology Hepatocytes - metabolism Hepatocytes - virology HIV HIV infections HIV Infections - metabolism HIV Infections - pathology HIV Infections - virology HIV Long Terminal Repeat - genetics HIV-1 - physiology Human immunodeficiency virus Humans Infection Infections Infectious diseases Inflammation Internal medicine Liver Long terminal repeat Lymphocytes Medical research Medicine NF-kappa B - genetics NF-kappa B - metabolism NF-κB protein Pathogenesis Penicillin Plasmids Promoter Regions, Genetic - genetics Proteins Replication tat Gene Products, Human Immunodeficiency Virus - genetics tat Gene Products, Human Immunodeficiency Virus - metabolism Tat protein Transcription (Genetics) Transcription activation Tumor Necrosis Factor-alpha - pharmacology Tumor necrosis factor-TNF Tumor necrosis factor-α Viral Core Proteins - genetics Viral Core Proteins - metabolism Virions Virology Virus Replication Viruses
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