Psychoactive pharmaceuticals induce fish gene expression profiles associated with human idiopathic autism

Idiopathic autism, caused by genetic susceptibility interacting with unknown environmental triggers, has increased dramatically in the past 25 years. Identifying environmental triggers has been difficult due to poorly understood pathophysiology and subjective definitions of autism. The use of antide...

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Veröffentlicht in:	PloS one 2012-06, Vol.7 (6), p.e32917
Hauptverfasser:	Thomas, Michael A, Klaper, Rebecca D
Format:	Artikel
Sprache:	eng
Schlagworte:	Analysis Animals Antidepressants Autism Autistic Disorder - chemically induced Autistic Disorder - genetics Autistic Disorder - metabolism Behavior Biology Biosynthesis Bipolar disorder Brain Carbamazepine Carbamazepine - toxicity Cell Adhesion Molecules - metabolism Contamination Cyclohexanols - toxicity Cyprinidae - genetics Cyprinidae - metabolism Disease susceptibility DNA microarrays Drinking water Drugs Fetuses Fish Fishes Fluoxetine Fluoxetine - toxicity Gene expression Gene Expression Profiling Gene Expression Regulation - drug effects Genes Genetic aspects Humans Medicine Mental disorders Microarray Analysis Multiple sclerosis Nerve Tissue Proteins - metabolism Nervous system diseases Neurological diseases Neurological disorders Parkinson's disease Pharmaceuticals Pregnancy Pregnant women Psychiatry Psychotropic Drugs - toxicity Receptors, Immunologic - metabolism Schizophrenia Serotonin Venlafaxine Venlafaxine Hydrochloride Water Pollutants, Chemical - toxicity Womens health
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