lin-28 controls the succession of cell fate choices via two distinct activities

lin-28 is a conserved regulator of cell fate succession in animals. In Caenorhabditis elegans, it is a component of the heterochronic gene pathway that governs larval developmental timing, while its vertebrate homologs promote pluripotency and control differentiation in diverse tissues. The RNA bind...

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Veröffentlicht in:PLoS genetics 2012-03, Vol.8 (3), p.e1002588-e1002588
Hauptverfasser: Vadla, Bhaskar, Kemper, Kevin, Alaimo, Jennifer, Heine, Christian, Moss, Eric G
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Kemper, Kevin
Alaimo, Jennifer
Heine, Christian
Moss, Eric G
description lin-28 is a conserved regulator of cell fate succession in animals. In Caenorhabditis elegans, it is a component of the heterochronic gene pathway that governs larval developmental timing, while its vertebrate homologs promote pluripotency and control differentiation in diverse tissues. The RNA binding protein encoded by lin-28 can directly inhibit let-7 microRNA processing by a novel mechanism that is conserved from worms to humans. We found that C. elegans LIN-28 protein can interact with four distinct let-7 family pre-microRNAs, but in vivo inhibits the premature accumulation of only let-7. Surprisingly, however, lin-28 does not require let-7 or its relatives for its characteristic promotion of second larval stage cell fates. In other words, we find that the premature accumulation of mature let-7 does not account for lin-28's precocious phenotype. To explain let-7's role in lin-28 activity, we provide evidence that lin-28 acts in two steps: first, the let-7-independent positive regulation of hbl-1 through its 3'UTR to control L2 stage-specific cell fates; and second, a let-7-dependent step that controls subsequent fates via repression of lin-41. Our evidence also indicates that let-7 functions one stage earlier in C. elegans development than previously thought. Importantly, lin-28's two-step mechanism resembles that of the heterochronic gene lin-14, and the overlap of their activities suggests a clockwork mechanism for developmental timing. Furthermore, this model explains the previous observation that mammalian Lin28 has two genetically separable activities. Thus, lin-28's two-step mechanism may be an essential feature of its evolutionarily conserved role in cell fate succession.
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This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited: Vadla B, Kemper K, Alaimo J, Heine C, Moss EG (2012) lin-28 Controls the Succession of Cell Fate Choices via Two Distinct Activities. 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subjects 3' Untranslated Regions - genetics
Animals
Bacterial genetics
Binding proteins
Biology
Caenorhabditis elegans
Caenorhabditis elegans - genetics
Caenorhabditis elegans - growth & development
Caenorhabditis elegans Proteins - genetics
Caenorhabditis elegans Proteins - metabolism
Cell Differentiation - genetics
DNA-Binding Proteins - genetics
DNA-Binding Proteins - metabolism
Gene Expression Regulation, Developmental
Genes
Genetic aspects
Genetics
Larva - genetics
Larva - growth & development
MicroRNAs
Nematodes
Nuclear Proteins - genetics
Nuclear Proteins - metabolism
Phenotype
Physiological aspects
Proteins
Repressor Proteins - genetics
RNA
Transcription Factors - genetics
Transcription Factors - metabolism
title lin-28 controls the succession of cell fate choices via two distinct activities
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