Modulation of calcium-dependent inactivation of L-type Ca2+ channels via β-adrenergic signaling in thalamocortical relay neurons

Neuronal high-voltage-activated (HVA) Ca(2+) channels are rapidly inactivated by a mechanism that is termed Ca(2+)-dependent inactivation (CDI). In this study we have shown that β-adrenergic receptor (βAR) stimulation inhibits CDI in rat thalamocortical (TC) relay neurons. This effect can be blocked...

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Veröffentlicht in:PloS one 2011-12, Vol.6 (12), p.e27474
Hauptverfasser: Rankovic, Vladan, Landgraf, Peter, Kanyshkova, Tatyana, Ehling, Petra, Meuth, Sven G, Kreutz, Michael R, Budde, Thomas, Munsch, Thomas
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Sprache:eng
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