Altered Energy Homeostasis and Resistance to Diet-Induced Obesity in KRAP-Deficient Mice

Obesity and related metabolic disorders have become leading causes of adult morbidity and mortality. KRAP (Ki-ras-induced actin-interacting protein) is a cytoskeleton-associated protein and a ubiquitous protein among tissues, originally identified as a cancer-related molecule, however, its physiolog...

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Veröffentlicht in:	PloS one 2009-01, Vol.4 (1), p.e4240-e4240
Hauptverfasser:	Fujimoto, Takahiro, Miyasaka, Kyoko, Koyanagi, Midori, Tsunoda, Toshiyuki, Baba, Iwai, Doi, Keiko, Ohta, Minoru, Kato, Norihiro, Sasazuki, Takehiko, Shirasawa, Senji
Format:	Artikel
Sprache:	eng
Schlagworte:	Acetyl-CoA carboxylase Acetyl-CoA Carboxylase - metabolism Actin Adipocytes Adipose tissue Adipose tissue (brown) Adipose Tissue - metabolism Adiposity - genetics Animal Feed Animal tissues Animals binding proteins Biology Biosynthesis brown adipose tissue Cancer Cloning Cytoskeleton Diabetes Diabetes and Endocrinology/Obesity Diet Energy balance energy metabolism Fatty Acid Synthases - metabolism Fatty acid synthesis Fatty acids Fatty-acid synthase Food food intake Genes Gerontology Glucose Glucose - metabolism Glucose tolerance Health aspects High fat diet Homeostasis Hyperphagia Insulin Insulin - metabolism Insulin resistance Ki-ras-induced actin-interacting protein leptin lipid metabolism Liver Liver - metabolism Medicine Membrane Proteins - metabolism Metabolic disorders Metabolic rate Metabolism Mice Mice, Transgenic Microfilament Proteins - genetics Microfilament Proteins - physiology Models, Biological Molecular Biology Morbidity Mortality Muscle proteins Nutrition/Obesity Obesity Obesity - genetics Obesity - pathology Physiological aspects Physiology Rodents Thyroid gland Type 2 diabetes
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