SOD3 reduces inflammatory cell migration by regulating adhesion molecule and cytokine expression

Inflammatory cell migration characteristic of ischemic damages has a dual role providing the tissue with factors needed for tissue injury recovery simultaneously causing deleterious development depending on the quality and the quantity of infiltrated cells. Extracellular superoxide dismutase (SOD3)...

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Veröffentlicht in:	PloS one 2009-06, Vol.4 (6), p.e5786-e5786
Hauptverfasser:	Laurila, Juha P, Laatikainen, Lilja E, Castellone, Maria D, Laukkanen, Mikko O
Format:	Artikel
Sprache:	eng
Schlagworte:	Adhesion Analysis Animal models Animals Apolipoproteins Cardiovascular Disorders/Peripheral Vascular Disease CD3 antigen Cell adhesion & migration Cell Adhesion Molecules - metabolism Cell Biology/Cell Adhesion Cell Line Cell Movement Cell proliferation Cellular signal transduction Chemokines Cloning Cooperation Cytokines Cytokines - biosynthesis Damage prevention E-selectin Extravasation Female Gene expression Granulocytes - cytology Humans Immunology Infiltration Inflammation Inflammatory diseases Injuries Interleukin 1 Interleukin 6 Ischemia Laboratories Lesions Leukemia Leukocyte migration Leukocytes (granulocytic) Lymphocytes T Macrophages Male Mice Mice, Inbred BALB C Models, Biological Monocyte chemoattractant protein 1 Oxygen P-selectin Peritonitis Proteins Rats Rats, Inbred F344 Reactive Oxygen Species - metabolism Recovery Signal processing Superoxide dismutase Superoxide Dismutase - biosynthesis Superoxide Dismutase - physiology Superoxides T cells Tissues Transcription factors Tumor necrosis factor-TNF Tumor necrosis factor-α Veins & arteries
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