CD4 T cell immunity is critical for the control of simian varicella virus infection in a nonhuman primate model of VZV infection

Primary infection with varicella zoster virus (VZV) results in varicella (more commonly known as chickenpox) after which VZV establishes latency in sensory ganglia. VZV can reactivate to cause herpes zoster (shingles), a debilitating disease that affects one million individuals in the US alone annua...

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Veröffentlicht in:	PLoS pathogens 2011-11, Vol.7 (11), p.e1002367-e1002367
Hauptverfasser:	Haberthur, Kristen, Engelmann, Flora, Park, Byng, Barron, Alex, Legasse, Alfred, Dewane, Jesse, Fischer, Miranda, Kerns, Amelia, Brown, Monica, Messaoudi, Ilhem
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Antigens, CD20 - immunology B-Lymphocytes - immunology CD4 Antigens - immunology CD4-Positive T-Lymphocytes - immunology CD8-Positive T-Lymphocytes - immunology Chickenpox - immunology Chickenpox - prevention & control Chickenpox - virology Chickenpox Vaccine - immunology Disease Models, Animal Health aspects Herpesviridae Infections - immunology Herpesviridae Infections - virology Herpesvirus 3, Human - immunology Immune response Immunology Infections Macaca mulatta Medicine Physiological aspects T cell receptors T cells Vaccines Varicella-zoster virus Varicellovirus - immunology Varicellovirus - physiology Viral Load Virus Replication
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