An erythrocyte vesicle protein exported by the malaria parasite promotes tubovesicular lipid import from the host cell surface

Plasmodium falciparum is the protozoan parasite that causes the most virulent of human malarias. The blood stage parasites export several hundred proteins into their host erythrocyte that underlie modifications linked to major pathologies of the disease and parasite survival in the blood. Unfortunat...

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Veröffentlicht in:	PLoS pathogens 2008-08, Vol.4 (8), p.e1000118-e1000118
Hauptverfasser:	Tamez, Pamela A, Bhattacharjee, Souvik, van Ooij, Christiaan, Hiller, N Luisa, Llinás, Manuel, Balu, Bharath, Adams, John H, Haldar, Kasturi
Format:	Artikel
Sprache:	eng
Schlagworte:	Animals Biological Transport - genetics Cell Biology/Membranes and Sorting Erythrocytes Erythrocytes - metabolism Erythrocytes - parasitology Gene Expression Regulation - genetics Genetics and Genomics/Gene Expression Genetics and Genomics/Genomics Genome, Protozoan - genetics Humans Infectious Diseases/Protozoal Infections Infectious Diseases/Tropical and Travel-Associated Diseases Intracellular Membranes - metabolism Intracellular Membranes - parasitology Lipid Metabolism - genetics Lipids - genetics Malaria Membrane Proteins - genetics Membrane Proteins - metabolism Microbiology/Parasitology Parasites Plasmodium falciparum Plasmodium falciparum - genetics Plasmodium falciparum - metabolism Proteins Protozoan Proteins - genetics Protozoan Proteins - metabolism
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