Endothelial NO/cGMP/VASP Signaling Attenuates Kupffer Cell Activation and Hepatic Insulin Resistance Induced by High-Fat Feeding

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Veröffentlicht in:Diabetes (New York, N.Y.) N.Y.), 2011, Vol.60 (11), p.2192-2201
Hauptverfasser: TATEYA, Sanshiro, RIZZO, Norma O, HANDA, Priya, CHENG, Andrew M, MORGAN-STEVENSON, Vicki, DAUM, Guenter, CLOWES, Alexander W, MORTON, Gregory J, SCHWARTZ, Michael W, KIM, Francis
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container_title Diabetes (New York, N.Y.)
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creator TATEYA, Sanshiro
RIZZO, Norma O
HANDA, Priya
CHENG, Andrew M
MORGAN-STEVENSON, Vicki
DAUM, Guenter
CLOWES, Alexander W
MORTON, Gregory J
SCHWARTZ, Michael W
KIM, Francis
description
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ispartof Diabetes (New York, N.Y.), 2011, Vol.60 (11), p.2192-2201
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source Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals; Journals@Ovid Complete; PubMed Central
subjects Biological and medical sciences
Diabetes. Impaired glucose tolerance
Endocrine pancreas. Apud cells (diseases)
Endocrinopathies
Etiopathogenesis. Screening. Investigations. Target tissue resistance
Medical sciences
title Endothelial NO/cGMP/VASP Signaling Attenuates Kupffer Cell Activation and Hepatic Insulin Resistance Induced by High-Fat Feeding
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