Oligomers of β-amyloid are sequestered into and seed new plaques in the brains of an AD mouse model : Beta-amylid and tau protein abnormalities in Alzheimer's disease
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Veröffentlicht in: | Experimental neurology 2010, Vol.223 (2), p.394-400 |
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container_title | Experimental neurology |
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creator | GASPAR, Renee C VILLARREAL, Stephanie A BOWLES, Nicole HEPLER, Robert W JOYCE, Joseph G SHUGHRUE, Paul J |
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ispartof | Experimental neurology, 2010, Vol.223 (2), p.394-400 |
issn | 0014-4886 1090-2430 |
language | eng |
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source | Elsevier ScienceDirect Journals |
subjects | Biological and medical sciences Degenerative and inherited degenerative diseases of the nervous system. Leukodystrophies. Prion diseases Medical sciences Neurology |
title | Oligomers of β-amyloid are sequestered into and seed new plaques in the brains of an AD mouse model : Beta-amylid and tau protein abnormalities in Alzheimer's disease |
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