Amphotericin B-Induced Renal Tubular Cell Injury Is Mediated by Na+ Influx through Ion-Permeable Pores and Subsequent Activation of Mitogen-Activated Protein Kinases and Elevation of Intracellular Ca2+ Concentration
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| Veröffentlicht in: | Antimicrobial Agents and Chemotherapy 2009-04, Vol.53 (4), p.1420-1426 |
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| container_title | Antimicrobial Agents and Chemotherapy |
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| creator | YANO, Takahisa ITOH, Yoshinori KAWAMURA, Eiko MAEDA, Asuka EGASHIRA, Nobuaki NISHIDA, Motohiro KUROSE, Hitoshi OISHI, Ryozo |
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| doi_str_mv | 10.1128/aac.01137-08 |
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</description><identifier>ISSN: 0066-4804</identifier><identifier>EISSN: 1098-6596</identifier><identifier>DOI: 10.1128/aac.01137-08</identifier><identifier>PMID: 19139282</identifier><identifier>CODEN: AACHAX</identifier><language>eng</language><publisher>Washington, DC: American Society for Microbiology</publisher><subject>Adenosine Triphosphate - analysis ; Amphotericin B ; Amphotericin B - toxicity ; Animals ; Antibiotics. Antiinfectious agents. Antiparasitic agents ; Antifungal Agents ; Antifungal Agents - toxicity ; Biological and medical sciences ; Calcium ; Calcium - metabolism ; Cholesterol - metabolism ; Endoplasmic Reticulum - drug effects ; Endoplasmic Reticulum - physiology ; Extracellular Signal-Regulated MAP Kinases - physiology ; Kidney Tubules ; Kidney Tubules - drug effects ; Kidney Tubules - pathology ; LLC-PK1 Cells ; MAP Kinase Signaling System ; MAP Kinase Signaling System - physiology ; Mechanisms of Action: Physiological Effects ; Medical sciences ; Mitochondria - drug effects ; Mitochondria - physiology ; Pharmacology. Drug treatments ; Sodium ; Sodium - metabolism ; Swine</subject><ispartof>Antimicrobial Agents and Chemotherapy, 2009-04, Vol.53 (4), p.1420-1426</ispartof><rights>2009 INIST-CNRS</rights><rights>Copyright © 2009 American Society for Microbiology</rights><rights>Copyright © 2009, American Society for Microbiology</rights><lds50>peer_reviewed</lds50><oa>free_for_read</oa><woscitedreferencessubscribed>false</woscitedreferencessubscribed></display><links><openurl>$$Topenurl_article</openurl><openurlfulltext>$$Topenurlfull_article</openurlfulltext><thumbnail>$$Tsyndetics_thumb_exl</thumbnail><linktopdf>$$Uhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC2663073/pdf/$$EPDF$$P50$$Gpubmedcentral$$H</linktopdf><linktohtml>$$Uhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC2663073/$$EHTML$$P50$$Gpubmedcentral$$H</linktohtml><link.rule.ids>230,314,723,776,780,881,27901,27902,53766,53768</link.rule.ids><backlink>$$Uhttp://pascal-francis.inist.fr/vibad/index.php?action=getRecordDetail&idt=21284254$$DView record in Pascal Francis$$Hfree_for_read</backlink><backlink>$$Uhttps://www.ncbi.nlm.nih.gov/pubmed/19139282$$D View this record in MEDLINE/PubMed$$Hfree_for_read</backlink></links><search><creatorcontrib>YANO, Takahisa</creatorcontrib><creatorcontrib>ITOH, Yoshinori</creatorcontrib><creatorcontrib>KAWAMURA, Eiko</creatorcontrib><creatorcontrib>MAEDA, Asuka</creatorcontrib><creatorcontrib>EGASHIRA, Nobuaki</creatorcontrib><creatorcontrib>NISHIDA, Motohiro</creatorcontrib><creatorcontrib>KUROSE, Hitoshi</creatorcontrib><creatorcontrib>OISHI, Ryozo</creatorcontrib><title>Amphotericin B-Induced Renal Tubular Cell Injury Is Mediated by Na+ Influx through Ion-Permeable Pores and Subsequent Activation of Mitogen-Activated Protein Kinases and Elevation of Intracellular Ca2+ Concentration</title><title>Antimicrobial Agents and Chemotherapy</title><addtitle>AAC</addtitle><addtitle>Antimicrob Agents Chemother</addtitle><description>Classifications
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</description><subject>Adenosine Triphosphate - analysis</subject><subject>Amphotericin B</subject><subject>Amphotericin B - toxicity</subject><subject>Animals</subject><subject>Antibiotics. Antiinfectious agents. 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Antiinfectious agents. Antiparasitic agents</topic><topic>Antifungal Agents</topic><topic>Antifungal Agents - toxicity</topic><topic>Biological and medical sciences</topic><topic>Calcium</topic><topic>Calcium - metabolism</topic><topic>Cholesterol - metabolism</topic><topic>Endoplasmic Reticulum - drug effects</topic><topic>Endoplasmic Reticulum - physiology</topic><topic>Extracellular Signal-Regulated MAP Kinases - physiology</topic><topic>Kidney Tubules</topic><topic>Kidney Tubules - drug effects</topic><topic>Kidney Tubules - pathology</topic><topic>LLC-PK1 Cells</topic><topic>MAP Kinase Signaling System</topic><topic>MAP Kinase Signaling System - physiology</topic><topic>Mechanisms of Action: Physiological Effects</topic><topic>Medical sciences</topic><topic>Mitochondria - drug effects</topic><topic>Mitochondria - physiology</topic><topic>Pharmacology. 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</abstract><cop>Washington, DC</cop><pub>American Society for Microbiology</pub><pmid>19139282</pmid><doi>10.1128/aac.01137-08</doi><tpages>7</tpages><oa>free_for_read</oa></addata></record> |
| fulltext | fulltext |
| identifier | ISSN: 0066-4804 |
| ispartof | Antimicrobial Agents and Chemotherapy, 2009-04, Vol.53 (4), p.1420-1426 |
| issn | 0066-4804 1098-6596 |
| language | eng |
| recordid | cdi_pascalfrancis_primary_21284254 |
| source | MEDLINE; Elektronische Zeitschriftenbibliothek - Frei zugängliche E-Journals; PubMed Central |
| subjects | Adenosine Triphosphate - analysis Amphotericin B Amphotericin B - toxicity Animals Antibiotics. Antiinfectious agents. Antiparasitic agents Antifungal Agents Antifungal Agents - toxicity Biological and medical sciences Calcium Calcium - metabolism Cholesterol - metabolism Endoplasmic Reticulum - drug effects Endoplasmic Reticulum - physiology Extracellular Signal-Regulated MAP Kinases - physiology Kidney Tubules Kidney Tubules - drug effects Kidney Tubules - pathology LLC-PK1 Cells MAP Kinase Signaling System MAP Kinase Signaling System - physiology Mechanisms of Action: Physiological Effects Medical sciences Mitochondria - drug effects Mitochondria - physiology Pharmacology. Drug treatments Sodium Sodium - metabolism Swine |
| title | Amphotericin B-Induced Renal Tubular Cell Injury Is Mediated by Na+ Influx through Ion-Permeable Pores and Subsequent Activation of Mitogen-Activated Protein Kinases and Elevation of Intracellular Ca2+ Concentration |
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