Indomethacin-induced radiosensitization and inhibition of ionizing radiation-induced NF-KB activation in HeLa cells occur via a mechanism involving p38 MAP kinase
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Veröffentlicht in: | Cancer research (Chicago, Ill.) Ill.), 2001, Vol.61 (20), p.7689-7696 |
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container_title | Cancer research (Chicago, Ill.) |
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creator | BRADBURY, C. Matthew MARKOVINA, Stephanie WEI, S. Jack RENE, Luis M ZOBERI, Imran HORIKOSHI, Nobuo GIUS, David |
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ispartof | Cancer research (Chicago, Ill.), 2001, Vol.61 (20), p.7689-7696 |
issn | 0008-5472 1538-7445 |
language | eng |
recordid | cdi_pascalfrancis_primary_14106468 |
source | American Association for Cancer Research; EZB-FREE-00999 freely available EZB journals |
subjects | Biological and medical sciences Biological effects of radiation Fundamental and applied biological sciences. Psychology Ionizing radiations Medical sciences Radiation therapy and radiosensitizing agent Tissues, organs and organisms biophysics Treatment with physical agents Treatment. General aspects Tumors |
title | Indomethacin-induced radiosensitization and inhibition of ionizing radiation-induced NF-KB activation in HeLa cells occur via a mechanism involving p38 MAP kinase |
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